Millipore

Anti-NR2B

#06-600 · western blot

80
Citing papers
3.2k
Combined citations
Supplier page Find this product

Research areas

Neuroscience and Neuropharmacology Research16Genetics and Neurodevelopmental Disorders9Neurogenesis and neuroplasticity mechanisms6Memory and Neural Mechanisms6Neurotransmitter Receptor Influence on Behavior5Genetic Neurodegenerative Diseases4Epigenetics and DNA Methylation3Ubiquitin and proteasome pathways3

Papers citing this product

1
Cellular and Synaptic Mechanisms of Anti-NMDA Receptor Encephalitis
Journal of Neuroscience·2010·1.1k cit.·DOI
2
Particulate air pollutants, APOE alleles and their contributions to cognitive impairment in older women and to amyloidogenesis in experimental models
Translational Psychiatry·2017·435 cit.·DOI
3
Inhibition of EHMT1/2 rescues synaptic and cognitive functions for Alzheimer’s disease
Brain·2019·174 cit.·DOI
4
Interaction of Postsynaptic Density Protein-95 with NMDA Receptors Influences Excitotoxicity in the Yeast Artificial Chromosome Mouse Model of Huntington's Disease
Journal of Neuroscience·2009·96 cit.·DOI
5
VCP and ATL1 regulate endoplasmic reticulum and protein synthesis for dendritic spine formation
Nature Communications·2016·83 cit.·DOI
6
Increasing the GluN2A/GluN2B Ratio in Neurons of the Mouse Basal and Lateral Amygdala Inhibits the Modification of an Existing Fear Memory Trace
Journal of Neuroscience·2016·78 cit.·DOI
7
The calcium sensor Copine-6 regulates spine structural plasticity and learning and memory
Nature Communications·2016·76 cit.·DOI
8
Disrupted Glutamatergic Transmission in Prefrontal Cortex Contributes to Behavioral Abnormality in an Animal Model of ADHD
Neuropsychopharmacology·2017·73 cit.·DOI
9
Chemogenetic Activation of Prefrontal Cortex Rescues Synaptic and Behavioral Deficits in a Mouse Model of 16p11.2 Deletion Syndrome
Journal of Neuroscience·2018·71 cit.·DOI
10
Histone deacetylase inhibitor MS-275 restores social and synaptic function in a Shank3-deficient mouse model of autism
Neuropsychopharmacology·2018·66 cit.·DOI

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