Proteintech

VCP antibody

#10736-1-AP · western blot · $449

66
Evidence Score · Well-Established
Used by 278 independent labs across 327 papers. How we score this →
23
Citing papers
427
Combined citations
Supplier page Find this product

Research areas

Endoplasmic Reticulum Stress and Disease9Ubiquitin and proteasome pathways5Cellular transport and secretion4Autophagy in Disease and Therapy4Genetics and Neurodevelopmental Disorders3Mitochondrial Function and Pathology3Ferroptosis and cancer prognosis2Glycosylation and Glycoproteins Research2

Papers citing this product

1
Berberine alleviates liver fibrosis through inducing ferrous redox to activate ROS-mediated hepatic stellate cells ferroptosis
Cell Death Discovery·2021·101 cit.·DOI
2
UBXD8 mediates mitochondria‐associated degradation to restrain apoptosis and mitophagy
EMBO Reports·2022·50 cit.·DOI
3
p97/VCP inhibition causes excessive MRE11-dependent DNA end resection promoting cell killing after ionizing radiation
Cell Reports·2021·45 cit.·DOI
4
VCP/p97-associated proteins are binders and debranching enzymes of K48–K63-branched ubiquitin chains
Nature Structural & Molecular Biology·2024·39 cit.·DOI
5
SEL1L-HRD1 interaction is required to form a functional HRD1 ERAD complex
Nature Communications·2024·37 cit.·DOI
6
Analysis of Zika virus capsid-Aedes aegypti mosquito interactome reveals pro-viral host factors critical for establishing infection
Nature Communications·2021·36 cit.·DOI
7
VPS13D interacts with VCP/p97 and negatively regulates ER- mitochondrial interactions
Molecular Biology of the Cell·2021·30 cit.·DOI
8
SPRTN protease-cleaved MRE11 decreases DNA repair and radiosensitises cancer cells
Cell Death and Disease·2021·18 cit.·DOI
9
HUWE1 Amplifies Ubiquitin Modifications to Broadly Stimulate Clearance of Proteins and Aggregates
bioRxiv (Cold Spring Harbor Laboratory)·2023·17 cit.·DOI
10
Comprehensive approach to study branched ubiquitin chains reveals roles for K48-K63 branches in VCP/p97-related processes
bioRxiv (Cold Spring Harbor Laboratory)·2023·16 cit.·DOI

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