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Cell Signaling Technology
Bora (D2B9) Rabbit Monoclonal Antibody
#12109 · $161
100
Citing papers
5.8k
Combined citations
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Research areas
Autophagy in Disease and Therapy
24
Calcium signaling and nucleotide metabolism
18
Muscle Physiology and Disorders
17
Extracellular vesicles in disease
7
Mitochondrial Function and Pathology
6
Mesenchymal stem cell research
6
Muscle metabolism and nutrition
5
Growth Hormone and Insulin-like Growth Factors
4
Papers citing this product
1
MCOLN1 is a ROS sensor in lysosomes that regulates autophagy
Nature Communications
·
2016
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549 cit.
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DOI
2
Activation of mitochondrial TUFM ameliorates metabolic dysregulation through coordinating autophagy induction
Communications Biology
·
2021
·
332 cit.
·
DOI
3
Mesenchymal stromal cell apoptosis is required for their therapeutic function
Nature Communications
·
2021
·
254 cit.
·
DOI
4
Sphingosylphosphorylcholine and Lysophosphatidylcholine Are Ligands for the G Protein-coupled Receptor GPR4
Journal of Biological Chemistry
·
2001
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241 cit.
·
DOI
5
Metformin induces cell cycle arrest, apoptosis and autophagy through ROS/JNK signaling pathway in human osteosarcoma
International Journal of Biological Sciences
·
2019
·
191 cit.
·
DOI
6
Rapid nontranscriptional activation of endothelial nitric oxide synthase mediates increased cerebral blood flow and stroke protection by corticosteroids
Journal of Clinical Investigation
·
2002
·
174 cit.
·
DOI
7
Insulin-Like Growth Factor-1 Receptor Is Regulated by microRNA-133 during Skeletal Myogenesis
PLoS ONE
·
2011
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166 cit.
·
DOI
8
Mitochondrial ROS-induced lysosomal dysfunction impairs autophagic flux and contributes to M1 macrophage polarization in a diabetic condition
Clinical Science
·
2019
·
153 cit.
·
DOI
9
TRIP13 promotes error-prone nonhomologous end joining and induces chemoresistance in head and neck cancer
Nature Communications
·
2014
·
152 cit.
·
DOI
10
Losing Control: Excessive Alcohol Seeking after Selective Inactivation of Cue-Responsive Neurons in the Infralimbic Cortex
Journal of Neuroscience
·
2015
·
149 cit.
·
DOI
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