Cell Signaling Technology

LIN28B Antibody

#5422 · $317

100

Citing papers

12.2k

Combined citations

Supplier page Find this product

Research areas

Caveolin-1 and cellular processes13 PI3K/AKT/mTOR signaling in cancer11 Tuberous Sclerosis Complex Research8 Metabolism, Diabetes, and Cancer7 Ubiquitin and proteasome pathways7 Autophagy in Disease and Therapy6 Genetics and Neurodevelopmental Disorders5 MicroRNA in disease regulation5

Papers citing this product

1

Mutations causing syndromic autism define an axis of synaptic pathophysiology

Nature·2011·598 cit.·DOI

2

Response of a Neuronal Model of Tuberous Sclerosis to Mammalian Target of Rapamycin (mTOR) Inhibitors: Effects on mTORC1 and Akt Signaling Lead to Improved Survival and Function

Journal of Neuroscience·2008·502 cit.·DOI

3

Pharmacological Inhibition of mTORC1 Suppresses Anatomical, Cellular, and Behavioral Abnormalities in Neural-Specific<i>Pten</i>Knock-Out Mice

Journal of Neuroscience·2009·439 cit.·DOI

4

Hypersensitivity to mGluR5 and ERK1/2 Leads to Excessive Protein Synthesis in the Hippocampus of a Mouse Model of Fragile X Syndrome

Journal of Neuroscience·2010·384 cit.·DOI

5

Akt Signals through the Mammalian Target of Rapamycin Pathway to Regulate CNS Myelination

Journal of Neuroscience·2009·321 cit.·DOI

6

Inhibition of MMP-2 and MMP-9 decreases cellular migration, and angiogenesis in in vitro models of retinoblastoma

BMC Cancer·2017·276 cit.·DOI

7

Metformin kills and radiosensitizes cancer cells and preferentially kills cancer stem cells

Scientific Reports·2012·275 cit.·DOI

8

Minocycline Alleviates Death of Oligodendrocytes by Inhibiting Pro-Nerve Growth Factor Production in Microglia after Spinal Cord Injury

Journal of Neuroscience·2007·270 cit.·DOI

9

Inhibition of the Mammalian Target of Rapamycin Signaling Pathway Suppresses Dentate Granule Cell Axon Sprouting in a Rodent Model of Temporal Lobe Epilepsy

Journal of Neuroscience·2009·228 cit.·DOI

10

Type I IFN Triggers RIG-I/TLR3/NLRP3-dependent Inflammasome Activation in Influenza A Virus Infected Cells

PLoS Pathogens·2013·226 cit.·DOI

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