Cell Signaling Technology

PPAR gamma (81B8) Rabbit Monoclonal Antibody

#2443 · western blot · $173,351

66

Evidence Score · Well-Established

Used by 463 independent labs across 506 papers. How we score this →

100

Citing papers

29k

Combined citations

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Research areas

Cancer Immunotherapy and Biomarkers35 Immune Cell Function and Interaction15 Immunotherapy and Immune Responses10 CAR-T cell therapy research10 Autophagy in Disease and Therapy8 Lung Cancer Treatments and Mutations7 Immune cells in cancer6 Phagocytosis and Immune Regulation5

Papers citing this product

1

PD-1 Blockade with Nivolumab in Relapsed or Refractory Hodgkin's Lymphoma

New England Journal of Medicine·2014·3.5k cit.·DOI

2

VEGF-A modulates expression of inhibitory checkpoints on CD8+ T cells in tumors

The Journal of Experimental Medicine·2015·1.1k cit.·DOI

3

Quantitative Assessment of the Heterogeneity of PD-L1 Expression in Non–Small-Cell Lung Cancer

JAMA Oncology·2015·834 cit.·DOI

4

Dietary tryptophan metabolite released by intratumoral Lactobacillus reuteri facilitates immune checkpoint inhibitor treatment

Cell·2023·635 cit.·DOI

5

Host expression of PD-L1 determines efficacy of PD-L1 pathway blockade–mediated tumor regression

Journal of Clinical Investigation·2018·598 cit.·DOI

6

Melanoma-specific MHC-II expression represents a tumour-autonomous phenotype and predicts response to anti-PD-1/PD-L1 therapy

Nature Communications·2016·572 cit.·DOI

7

Improved antitumor activity of immunotherapy with BRAF and MEK inhibitors in <i>BRAF</i> <sup> <i>V600E</i> </sup> melanoma

Science Translational Medicine·2015·566 cit.·DOI

8

Cancer-associated fibroblasts induce antigen-specific deletion of CD8 + T Cells to protect tumour cells

Nature Communications·2018·558 cit.·DOI

9

Valosin-containing protein (VCP) is required for autophagy and is disrupted in VCP disease

The Journal of Cell Biology·2009·489 cit.·DOI

10

The Activation of MAPK in Melanoma Cells Resistant to BRAF Inhibition Promotes PD-L1 Expression That Is Reversible by MEK and PI3K Inhibition

Clinical Cancer Research·2012·470 cit.·DOI

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