Cell Signaling Technology

MMP-2 (D4M2N) Rabbit Monoclonal Antibody (BSA and Azide Free)

#35814 · $865

12
Citing papers
260
Combined citations
Supplier page Find this product

Research areas

PI3K/AKT/mTOR signaling in cancer3RNA Research and Splicing2Adipokines, Inflammation, and Metabolic Diseases2Autophagy in Disease and Therapy1Cellular transport and secretion1DNA and Nucleic Acid Chemistry1Cancer-related Molecular Pathways1DNA Repair Mechanisms1

Papers citing this product

1
Sestrin 2 confers primary resistance to sorafenib by simultaneously activating AKT and AMPK in hepatocellular carcinoma
Cancer Medicine·2018·50 cit.·DOI
2
Sphingolipids: the nexus between Gaucher disease and insulin resistance
Lipids in Health and Disease·2010·45 cit.·DOI
3
Transcriptional Regulation of an Insulin-Sensitizing Adipokine Adipolin/CTRP12 in Adipocytes by Krüppel-Like Factor 15
PLoS ONE·2013·42 cit.·DOI
4
Calycosin induces autophagy and apoptosis via Sestrin2/AMPK/mTOR in human papillary thyroid cancer cells
Frontiers in Pharmacology·2022·35 cit.·DOI
5
Insulin regulates the novel adipokine adipolin/CTRP12: in vivo and ex vivo effects
Journal of Endocrinology·2014·33 cit.·DOI
6
Tumor Suppressive Function of NQO1 in Cutaneous Squamous Cell Carcinoma (SCC) Cells
BioMed Research International·2019·27 cit.·DOI
7
The deletion of the protein phosphatase 1 regulator NIPP1 in testis causes hyperphosphorylation and degradation of the histone methyltransferase EZH2
Journal of Biological Chemistry·2018·15 cit.·DOI
8
Molecular mechanism of bis(maltolato)oxovanadium(IV)-induced insulin signaling in 3T3-L1 and IM9 cells: impact of dexamethasone
Journal of Molecular Endocrinology·2007·6 cit.·DOI
9
<scp>DNA</scp> damage‐induced allosteric activation of protein phosphatase <scp>PP1</scp> : <scp>NIPP1</scp> through Src kinase‐induced circularization of <scp>NIPP1</scp>
FEBS Journal·2024·5 cit.·DOI
10
Inhibition of SF3B1 affects recruitment of P-TEFb to chromatin through multiple mechanisms
bioRxiv (Cold Spring Harbor Laboratory)·2024·2 cit.·DOI

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