Cell Signaling Technology

SHP-1 (C14H6) Rabbit Monoclonal Antibody

#3759 · $156

100
Citing papers
11.5k
Combined citations
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Research areas

Cancer, Hypoxia, and Metabolism11TGF-β signaling in diseases11RNA modifications and cancer7Metabolism, Diabetes, and Cancer7Immune Cell Function and Interaction7Neurogenesis and neuroplasticity mechanisms6Cell Adhesion Molecules Research6MicroRNA in disease regulation6

Papers citing this product

1
A targetable LIFR−NF-κB−LCN2 axis controls liver tumorigenesis and vulnerability to ferroptosis
Nature Communications·2021·336 cit.·DOI
2
Replication gaps are a key determinant of PARP inhibitor synthetic lethality with BRCA deficiency
Molecular Cell·2021·315 cit.·DOI
3
Angiogenic factors FGF2 and PDGF-BB synergistically promote murine tumor neovascularization and metastasis
Journal of Clinical Investigation·2007·284 cit.·DOI
4
Transforming Growth Factor-β (TGF-β) Directly Activates the JAK1-STAT3 Axis to Induce Hepatic Fibrosis in Coordination with the SMAD Pathway
Journal of Biological Chemistry·2017·264 cit.·DOI
5
Xanthine oxidoreductase regulates macrophage IL1β secretion upon NLRP3 inflammasome activation
Nature Communications·2015·263 cit.·DOI
6
Tumour extracellular vesicles and particles induce liver metabolic dysfunction
Nature·2023·255 cit.·DOI
7
ROS-induced DNA damage and PARP-1 are required for optimal induction of starvation-induced autophagy
Cell Research·2012·243 cit.·DOI
8
β3Integrin and Src facilitate transforming growth factor-β mediated induction of epithelial-mesenchymal transition in mammary epithelial cells
Breast Cancer Research·2006·236 cit.·DOI
9
Role of Rho/ROCK and p38 MAP Kinase Pathways in Transforming Growth Factor-β-mediated Smad-dependent Growth Inhibition of Human Breast Carcinoma Cells in Vivo
Journal of Biological Chemistry·2004·224 cit.·DOI
10
Mitochondrial dysfunction in some triple-negative breast cancer cell lines: role of mTOR pathway and therapeutic potential
Breast Cancer Research·2014·218 cit.·DOI

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SHP-1 (C14H6) Rabbit Monoclonal Antibody — Evidence — PubIndex