Cell Signaling Technology

Phospho-FANCD2 (Ser222) Antibody

#4945 · western blot

100
Citing papers
8k
Combined citations
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Research areas

Ubiquitin and proteasome pathways9Cell death mechanisms and regulation7Cancer Cells and Metastasis6PI3K/AKT/mTOR signaling in cancer6Autophagy in Disease and Therapy6Cancer-related molecular mechanisms research5Pancreatic and Hepatic Oncology Research5MicroRNA in disease regulation5

Papers citing this product

1
Resveratrol inhibits phorbol ester-induced expression of COX-2 and activation of NF-κB in mouse skin by blocking IκB kinase activity
Carcinogenesis·2006·266 cit.·DOI
2
Oncogenic KRAS supports pancreatic cancer through regulation of nucleotide synthesis
Nature Communications·2018·266 cit.·DOI
3
miR-122 promotes hepatic lipogenesis via inhibiting the LKB1/AMPK pathway by targeting Sirt1 in non-alcoholic fatty liver disease
Molecular Medicine·2019·266 cit.·DOI
4
Personalizing Cancer Treatment in the Age of Global Genomic Analyses: <i>PALB2</i> Gene Mutations and the Response to DNA Damaging Agents in Pancreatic Cancer
Molecular Cancer Therapeutics·2010·246 cit.·DOI
5
EML4-ALK testing in non-small cell carcinomas of the lung: a review with recommendations
Archiv für Pathologische Anatomie und Physiologie und für Klinische Medicin·2012·211 cit.·DOI
6
PD-1 Increases PTEN Phosphatase Activity While Decreasing PTEN Protein Stability by Inhibiting Casein Kinase 2
Molecular and Cellular Biology·2013·186 cit.·DOI
7
MUC1 induces drug resistance in pancreatic cancer cells via upregulation of multidrug resistance genes
Oncogenesis·2013·174 cit.·DOI
8
Loss of NDRG2 expression activates PI3K-AKT signalling via PTEN phosphorylation in ATLL and other cancers
Nature Communications·2014·162 cit.·DOI
9
Resveratrol inhibits estrogen-induced breast carcinogenesis through induction of NRF2-mediated protective pathways
Carcinogenesis·2014·155 cit.·DOI
10
Evolutionary origins and interactomes of human, young microproteins and small peptides translated from short open reading frames
Molecular Cell·2023·153 cit.·DOI

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