Cell Signaling Technology

Phospho-Akt (Ser473) Antibody

#9271 · $150,863

66
Evidence Score · Well-Established
Used by 1,805 independent labs across 2,013 papers. How we score this →
100
Citing papers
28k
Combined citations
Supplier page Find this product

Research areas

PI3K/AKT/mTOR signaling in cancer14Autophagy in Disease and Therapy11MicroRNA in disease regulation7Adipokines, Inflammation, and Metabolic Diseases7Adipose Tissue and Metabolism7Pancreatic function and diabetes6Signaling Pathways in Disease6Extracellular vesicles in disease5

Papers citing this product

1
Systematic RNA interference reveals that oncogenic KRAS-driven cancers require TBK1
Nature·2009·4.2k cit.·DOI
2
Oncogenic Transformation by Inhibitor-Sensitive and -Resistant EGFR Mutants
PLoS Medicine·2005·683 cit.·DOI
3
Hypoxic Preconditioning Results in Increased Motility and Improved Therapeutic Potential of Human Mesenchymal Stem Cells
Stem Cells·2008·678 cit.·DOI
4
Circulating tumour DNA profiling reveals heterogeneity of EGFR inhibitor resistance mechanisms in lung cancer patients
Nature Communications·2016·657 cit.·DOI
5
Modulation of mammalian life span by the short isoform of p53
Genes & Development·2004·578 cit.·DOI
6
The short-chain fatty acid pentanoate suppresses autoimmunity by modulating the metabolic-epigenetic crosstalk in lymphocytes
Nature Communications·2019·509 cit.·DOI
7
Activin A Efficiently Specifies Definitive Endoderm from Human Embryonic Stem Cells Only When Phosphatidylinositol 3-Kinase Signaling Is Suppressed
Stem Cells·2007·475 cit.·DOI
8
Regulation of Embryonic Stem Cell Self-renewal by Phosphoinositide 3-Kinase-dependent Signaling
Journal of Biological Chemistry·2004·414 cit.·DOI
9
Baicalin ameliorates neuroinflammation-induced depressive-like behavior through inhibition of toll-like receptor 4 expression via the PI3K/AKT/FoxO1 pathway
Journal of Neuroinflammation·2019·407 cit.·DOI
10
Supermeres are functional extracellular nanoparticles replete with disease biomarkers and therapeutic targets
Nature Cell Biology·2021·399 cit.·DOI

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