Cell Signaling Technology

IRF-3 (D9J5Q) Mouse Monoclonal Antibody

#10949 · $158

100
Citing papers
4.3k
Combined citations
Supplier page Find this product

Research areas

Endoplasmic Reticulum Stress and Disease10MicroRNA in disease regulation10Autophagy in Disease and Therapy8interferon and immune responses8RNA Research and Splicing8Extracellular vesicles in disease7Neuroinflammation and Neurodegeneration Mechanisms6Hepatitis C virus research6

Papers citing this product

1
NAD <sup>+</sup> supplementation normalizes key Alzheimer’s features and DNA damage responses in a new AD mouse model with introduced DNA repair deficiency
Proceedings of the National Academy of Sciences·2018·457 cit.·DOI
2
Mitochondria-specific drug release and reactive oxygen species burst induced by polyprodrug nanoreactors can enhance chemotherapy
Nature Communications·2019·434 cit.·DOI
3
A rigorous method to enrich for exosomes from brain tissue
Journal of Extracellular Vesicles·2017·327 cit.·DOI
4
An evolutionary NS1 mutation enhances Zika virus evasion of host interferon induction
Nature Communications·2018·324 cit.·DOI
5
Antagonistic interplay between hypocretin and leptin in the lateral hypothalamus regulates stress responses
Nature Communications·2015·175 cit.·DOI
6
Hypocretin/Orexin Neurons Contribute to Hippocampus-Dependent Social Memory and Synaptic Plasticity in Mice
Journal of Neuroscience·2013·155 cit.·DOI
7
Integrated stress response is critical for gemcitabine resistance in pancreatic ductal adenocarcinoma
Cell Death and Disease·2015·126 cit.·DOI
8
BRAF<sup>V600</sup>inhibition alters the microRNA cargo in the vesicular secretome of malignant melanoma cells
Proceedings of the National Academy of Sciences·2017·126 cit.·DOI
9
Mast cell exosomes promote lung adenocarcinoma cell proliferation – role of KIT-stem cell factor signaling
Cell Communication and Signaling·2014·117 cit.·DOI
10
PTEN is a protein phosphatase that targets active PTK6 and inhibits PTK6 oncogenic signaling in prostate cancer
Nature Communications·2017·84 cit.·DOI

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