Cell Signaling Technology

Atg5 (D5F5U) Rabbit Monoclonal Antibody

#12994 · $149

100
Citing papers
7.6k
Combined citations
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Research areas

Autophagy in Disease and Therapy59Endoplasmic Reticulum Stress and Disease12MicroRNA in disease regulation7Epigenetics and DNA Methylation7Hippo pathway signaling and YAP/TAZ5Inflammasome and immune disorders5Cell death mechanisms and regulation5Sirtuins and Resveratrol in Medicine5

Papers citing this product

1
Aβ Oligomers Cause Localized Ca <sup>2+</sup> Elevation, Missorting of Endogenous Tau into Dendrites, Tau Phosphorylation, and Destruction of Microtubules and Spines
Journal of Neuroscience·2010·677 cit.·DOI
2
DUSP1 alleviates cardiac ischemia/reperfusion injury by suppressing the Mff-required mitochondrial fission and Bnip3-related mitophagy via the JNK pathways
Redox Biology·2017·459 cit.·DOI
3
High salt intake reprioritizes osmolyte and energy metabolism for body fluid conservation
Journal of Clinical Investigation·2017·199 cit.·DOI
4
Activation of the Unfolded Protein Response Is Required for Defenses against Bacterial Pore-Forming Toxin In Vivo
PLoS Pathogens·2008·194 cit.·DOI
5
Tumor-Stroma-Inflammation Networks Promote Pro-metastatic Chemokines and Aggressiveness Characteristics in Triple-Negative Breast Cancer
Frontiers in Immunology·2019·193 cit.·DOI
6
Transcriptional regulation of autophagy-lysosomal function in BRAF-driven melanoma progression and chemoresistance
Nature Communications·2019·171 cit.·DOI
7
Pore-forming toxins induce multiple cellular responses promoting survival
Cellular Microbiology·2011·151 cit.·DOI
8
The flavonoid 4,4′-dimethoxychalcone promotes autophagy-dependent longevity across species
Nature Communications·2019·139 cit.·DOI
9
Insulin-degrading enzyme secretion from astrocytes is mediated by an autophagy-based unconventional secretory pathway in Alzheimer disease
Autophagy·2016·129 cit.·DOI
10
Type IV Secretion-Dependent Activation of Host MAP Kinases Induces an Increased Proinflammatory Cytokine Response to Legionella pneumophila
PLoS Pathogens·2008·128 cit.·DOI

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