Cell Signaling Technology

JMJD1B (C69G2) Rabbit Monoclonal Antibody

#3314 · $161,351

64
Evidence Score · Well-Established
Used by 186 independent labs across 200 papers. How we score this →
100
Citing papers
10.7k
Combined citations
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Research areas

Lung Cancer Treatments and Mutations12PI3K/AKT/mTOR signaling in cancer11Cancer, Hypoxia, and Metabolism8Epigenetics and DNA Methylation7Cancer Cells and Metastasis6Lung Cancer Research Studies5Liver physiology and pathology5RNA Research and Splicing5

Papers citing this product

1
Intensive insulin therapy protects the endothelium of critically ill patients
Journal of Clinical Investigation·2005·466 cit.·DOI
2
A systems study reveals concurrent activation of AMPK and mTOR by amino acids
Nature Communications·2016·446 cit.·DOI
3
Anti-inflammatory effects of BHBA in both in vivo and in vitro Parkinson’s disease models are mediated by GPR109A-dependent mechanisms
Journal of Neuroinflammation·2015·262 cit.·DOI
4
TLR1- and TLR6-independent Recognition of Bacterial Lipopeptides
Journal of Biological Chemistry·2006·252 cit.·DOI
5
Sinusoidal Endothelial Dysfunction Precedes Inflammation and Fibrosis in a Model of NAFLD
PLoS ONE·2012·241 cit.·DOI
6
Blocking CDK1/PDK1/β-Catenin signaling by CDK1 inhibitor RO3306 increased the efficacy of sorafenib treatment by targeting cancer stem cells in a preclinical model of hepatocellular carcinoma
Theranostics·2018·234 cit.·DOI
7
Quantitative analysis of how Myc controls T cell proteomes and metabolic pathways during T cell activation
eLife·2020·218 cit.·DOI
8
EML4-ALK testing in non-small cell carcinomas of the lung: a review with recommendations
Archiv für Pathologische Anatomie und Physiologie und für Klinische Medicin·2012·211 cit.·DOI
9
Loss of the interleukin-6 receptor causes immunodeficiency, atopy, and abnormal inflammatory responses
The Journal of Experimental Medicine·2019·204 cit.·DOI
10
6-Phosphofructo-2-kinase (PFKFB3) promotes cell cycle progression and suppresses apoptosis via Cdk1-mediated phosphorylation of p27
Cell Death and Disease·2014·204 cit.·DOI

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