Cell Signaling Technology

Phospho-Thr-Pro-Pro Motif [pTPP] (D61C3) Rabbit Monoclonal Antibody

#5757 · $176,415

100

Citing papers

8.2k

Combined citations

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Research areas

Mitochondrial Function and Pathology14 Cell death mechanisms and regulation9 Autophagy in Disease and Therapy9 Endoplasmic Reticulum Stress and Disease7 Microtubule and mitosis dynamics7 DNA Repair Mechanisms6 T-cell and B-cell Immunology6 Epigenetics and DNA Methylation5

Papers citing this product

1

Mammalian TAK1 Activates Snf1 Protein Kinase in Yeast and Phosphorylates AMP-activated Protein Kinase in Vitro

Journal of Biological Chemistry·2006·461 cit.·DOI

2

Mitochondrial survivin inhibits apoptosis and promotes tumorigenesis

Journal of Clinical Investigation·2004·354 cit.·DOI

3

RTN1-C mediates cerebral ischemia/reperfusion injury via ER stress and mitochondria-associated apoptosis pathways

Cell Death and Disease·2017·270 cit.·DOI

4

Differential Expression of Apoptotic Protease-Activating Factor-1 and Caspase-3 Genes and Susceptibility to Apoptosis during Brain Development and after Traumatic Brain Injury

Journal of Neuroscience·2001·263 cit.·DOI

5

Omeprazole Blocks STAT6 Binding to the Eotaxin-3 Promoter in Eosinophilic Esophagitis Cells

PLoS ONE·2012·250 cit.·DOI

6

Phosphorylation of Synucleins by Members of the Polo-like Kinase Family

Journal of Biological Chemistry·2009·248 cit.·DOI

7

SOCS3-Mediated Blockade of JAK/STAT3 Signaling Pathway Reveals Its Major Contribution to Spinal Cord Neuroinflammation and Mechanical Allodynia after Peripheral Nerve Injury

Journal of Neuroscience·2010·207 cit.·DOI

8

Type I IFN Induces IL-10 Production in an IL-27–Independent Manner and Blocks Responsiveness to IFN-γ for Production of IL-12 and Bacterial Killing in <i>Mycobacterium tuberculosis</i> –Infected Macrophages

The Journal of Immunology·2014·195 cit.·DOI

9

Sulforaphane protects against rotenone-induced neurotoxicity in vivo: Involvement of the mTOR, Nrf2 and autophagy pathways

Scientific Reports·2016·171 cit.·DOI

10

Calpain Is a Major Cell Death Effector in Selective Striatal Degeneration Induced<i>In Vivo</i>by 3-Nitropropionate: Implications for Huntington's Disease

Journal of Neuroscience·2003·160 cit.·DOI

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