Cell Signaling Technology

Histone H4 (D2X4V) Rabbit Monoclonal Antibody (BSA and Azide Free)

#29672 · $865

18
Citing papers
479
Combined citations
Supplier page Find this product

Research areas

Ion Channels and Receptors4NF-κB Signaling Pathways3Signaling Pathways in Disease2Coagulation, Bradykinin, Polyphosphates, and Angioedema2Cancer therapeutics and mechanisms2Genomics, phytochemicals, and oxidative stress2Bioactive Compounds and Antitumor Agents2Cancer-related Molecular Pathways1

Papers citing this product

1
Cannabinoid-receptor-independent cell signalling by N-acylethanolamines
Biochemical Journal·2001·72 cit.·DOI
2
T-Lymphokine–Activated Killer Cell–Originated Protein Kinase Functions as a Positive Regulator of c-Jun-NH2-Kinase 1 Signaling and H-Ras–Induced Cell Transformation
Cancer Research·2007·63 cit.·DOI
3
Gain-of-function Mutations in Transient Receptor Potential C6 (TRPC6) Activate Extracellular Signal-regulated Kinases 1/2 (ERK1/2)
Journal of Biological Chemistry·2013·59 cit.·DOI
4
Redox effector factor-1, combined with reactive oxygen species, plays an important role in the transformation of JB6 cells
Carcinogenesis·2007·45 cit.·DOI
5
Absence of IQGAP1 Protein Leads to Insulin Resistance
Journal of Biological Chemistry·2017·33 cit.·DOI
6
IRS-1 Functions as a Molecular Scaffold to Coordinate IGF-I/IGFBP-2 Signaling During Osteoblast Differentiation
Journal of Bone and Mineral Research·2016·32 cit.·DOI
7
Volasertib preclinical activity in high-risk hepatoblastoma
Oncotarget·2019·31 cit.·DOI
8
Nerve Growth Factor Receptor TrkA, a New Receptor in Insulin Signaling Pathway in PC12 Cells
Journal of Biological Chemistry·2013·30 cit.·DOI
9
Cyclin D1 Induction by Benzo[a]pyrene-7,8-diol-9,10-epoxide via the Phosphatidylinositol 3-Kinase/Akt/MAPK- and p70s6k-dependent Pathway Promotes Cell Transformation and Tumorigenesis
Journal of Biological Chemistry·2009·30 cit.·DOI
10
Linoleic acid metabolite suppresses skin inflammation and tumor promotion in mice: possible roles of programmed cell death 4 induction
Carcinogenesis·2009·23 cit.·DOI

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