Cell Signaling Technology

PathScan® Total p53 Sandwich ELISA Kit

#7370 · $661

100
Citing papers
10.9k
Combined citations
Supplier page Find this product

Research areas

Advanced Glycation End Products research23Immune Response and Inflammation11Neuroinflammation and Neurodegeneration Mechanisms9Erythrocyte Function and Pathophysiology8Ubiquitin and proteasome pathways7Pain Mechanisms and Treatments7Protein Degradation and Inhibitors7Nerve injury and regeneration6

Papers citing this product

1
Membrane-associated Hsp72 from tumor-derived exosomes mediates STAT3-dependent immunosuppressive function of mouse and human myeloid-derived suppressor cells
Journal of Clinical Investigation·2010·985 cit.·DOI
2
Inhibition of Microglial Phagocytosis Is Sufficient To Prevent Inflammatory Neuronal Death
The Journal of Immunology·2011·372 cit.·DOI
3
Hepatic Ischemia/Reperfusion Injury Involves Functional TLR4 Signaling in Nonparenchymal Cells
The Journal of Immunology·2005·361 cit.·DOI
4
Role of Mitogen-Activated Protein Kinase Activation in Injured and Intact Primary Afferent Neurons for Mechanical and Heat Hypersensitivity after Spinal Nerve Ligation
Journal of Neuroscience·2004·310 cit.·DOI
5
Hemorrhagic Shock Induces NAD(P)H Oxidase Activation in Neutrophils: Role of HMGB1-TLR4 Signaling
The Journal of Immunology·2007·295 cit.·DOI
6
P2Y<sub>12</sub>Receptor Upregulation in Activated Microglia Is a Gateway of p38 Signaling and Neuropathic Pain
Journal of Neuroscience·2008·272 cit.·DOI
7
Mechanically activated ion channel PIEZO1 is required for lymphatic valve formation
Proceedings of the National Academy of Sciences·2018·265 cit.·DOI
8
CCAT1 is an enhancer-templated RNA that predicts BET sensitivity in colorectal cancer
Journal of Clinical Investigation·2016·230 cit.·DOI
9
MFG-E8 Mediates Primary Phagocytosis of Viable Neurons during Neuroinflammation
Journal of Neuroscience·2012·214 cit.·DOI
10
HMGB1 promotes the activation of NLRP3 and caspase-8 inflammasomes via NF-κB pathway in acute glaucoma
Journal of Neuroinflammation·2015·208 cit.·DOI

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