Cell Signaling Technology

Flotillin-2 (L294) Antibody

#3244 · $357

64
Evidence Score · Well-Established
Used by 186 independent labs across 199 papers. How we score this →
100
Citing papers
14.8k
Combined citations
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Research areas

PI3K/AKT/mTOR signaling in cancer6MicroRNA in disease regulation6Neuroscience and Neuropharmacology Research6Retinal Diseases and Treatments5Immune Response and Inflammation5Neuroinflammation and Neurodegeneration Mechanisms5Autophagy in Disease and Therapy5RNA Research and Splicing5

Papers citing this product

1
ER stress (PERK/eIF2α phosphorylation) mediates the polyglutamine-induced LC3 conversion, an essential step for autophagy formation
Cell Death and Differentiation·2006·923 cit.·DOI
2
Epigenetic regulation of the alternatively activated macrophage phenotype
Blood·2009·491 cit.·DOI
3
SOCS3 Deficiency Promotes M1 Macrophage Polarization and Inflammation
The Journal of Immunology·2012·464 cit.·DOI
4
Motif module map reveals enforcement of aging by continual NF-κB activity
Genes & Development·2007·454 cit.·DOI
5
Gli1 identifies osteogenic progenitors for bone formation and fracture repair
Nature Communications·2017·353 cit.·DOI
6
Enhanced PIP3 signaling in POMC neurons causes KATP channel activation and leads to diet-sensitive obesity
Journal of Clinical Investigation·2006·322 cit.·DOI
7
PLX4032, a selective BRAF <sup>V600E</sup> kinase inhibitor, activates the ERK pathway and enhances cell migration and proliferation of BRAF <sup>WT</sup> melanoma cells
Pigment Cell & Melanoma Research·2010·322 cit.·DOI
8
Entrectinib, a Pan–TRK, ROS1, and ALK Inhibitor with Activity in Multiple Molecularly Defined Cancer Indications
Molecular Cancer Therapeutics·2016·312 cit.·DOI
9
Mitochondrial oxidative stress caused by Sod2 deficiency promotes cellular senescence and aging phenotypes in the skin
Aging·2012·280 cit.·DOI
10
Differential roles of the ubiquitin proteasome system (UPS) and autophagy in the clearance of soluble and aggregated TDP-43 species
Journal of Cell Science·2014·267 cit.·DOI

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