Cell Signaling Technology

Anti-rabbit IgG F(ab')2 Fragment (Sepharose Bead Conjugate)

#6990 · $176

100
Citing papers
8.6k
Combined citations
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Research areas

Neuroscience and Neuropharmacology Research12Alzheimer's disease research and treatments10Mitochondrial Function and Pathology8Neuroinflammation and Neurodegeneration Mechanisms6Histone Deacetylase Inhibitors Research5Immune Cell Function and Interaction5Immune Response and Inflammation5Protease and Inhibitor Mechanisms5

Papers citing this product

1
Mitochondria-specific drug release and reactive oxygen species burst induced by polyprodrug nanoreactors can enhance chemotherapy
Nature Communications·2019·434 cit.·DOI
2
Anesthesia Leads to Tau Hyperphosphorylation through Inhibition of Phosphatase Activity by Hypothermia
Journal of Neuroscience·2007·389 cit.·DOI
3
Neuroprotective Effects of Regulators of the Glycogen Synthase Kinase-3β Signaling Pathway in a Transgenic Model of Alzheimer's Disease Are Associated with Reduced Amyloid Precursor Protein Phosphorylation
Journal of Neuroscience·2007·287 cit.·DOI
4
Acute Suppression of Spontaneous Neurotransmission Drives Synaptic Potentiation
Journal of Neuroscience·2013·276 cit.·DOI
5
Full Reversal of Alzheimer's Disease-Like Phenotype in a Mouse Model with Conditional Overexpression of Glycogen Synthase Kinase-3
Journal of Neuroscience·2006·253 cit.·DOI
6
Characterization of LINE-1 Ribonucleoprotein Particles
PLoS Genetics·2010·245 cit.·DOI
7
Insulin Dysfunction Induces<i>In Vivo</i>Tau Hyperphosphorylation through Distinct Mechanisms
Journal of Neuroscience·2007·236 cit.·DOI
8
Rapamycin Attenuates the Progression of Tau Pathology in P301S Tau Transgenic Mice
PLoS ONE·2013·225 cit.·DOI
9
Synergistic Neuroprotective Effects of Lithium and Valproic Acid or Other Histone Deacetylase Inhibitors in Neurons: Roles of Glycogen Synthase Kinase-3 Inhibition
Journal of Neuroscience·2008·214 cit.·DOI
10
Tissue- and cell-type–specific manifestations of heteroplasmic mtDNA 3243A&gt;G mutation in human induced pluripotent stem cell-derived disease model
Proceedings of the National Academy of Sciences·2013·204 cit.·DOI

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