Cell Signaling Technology

RIP3 (D4G2A) Rabbit Monoclonal Antibody

#95702 · $161

60
Citing papers
2.1k
Combined citations
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Research areas

Cell death mechanisms and regulation19Inflammasome and immune disorders12interferon and immune responses7Mitochondrial Function and Pathology6Phagocytosis and Immune Regulation4Endoplasmic Reticulum Stress and Disease4Drug-Induced Hepatotoxicity and Protection4Ferroptosis and cancer prognosis3

Papers citing this product

1
Identification of the PANoptosome: A Molecular Platform Triggering Pyroptosis, Apoptosis, and Necroptosis (PANoptosis)
Frontiers in Cellular and Infection Microbiology·2020·530 cit.·DOI
2
Ferroptosis driven by radical oxidation of n-6 polyunsaturated fatty acids mediates acetaminophen-induced acute liver failure
Cell Death and Disease·2020·335 cit.·DOI
3
RIPK1 Distinctly Regulates <i>Yersinia</i>-Induced Inflammatory Cell Death, PANoptosis
ImmunoHorizons·2020·198 cit.·DOI
4
GSK872 and necrostatin-1 protect retinal ganglion cells against necroptosis through inhibition of RIP1/RIP3/MLKL pathway in glutamate-induced retinal excitotoxic model of glaucoma
Journal of Neuroinflammation·2022·127 cit.·DOI
5
Necroptosis and Apoptosis Contribute to Cisplatin and Aminoglycoside Ototoxicity
Journal of Neuroscience·2019·66 cit.·DOI
6
Blocking reverse electron transfer-mediated mitochondrial DNA oxidation rescues cells from PANoptosis
Acta Pharmacologica Sinica·2023·63 cit.·DOI
7
Prolonged hypoxia alleviates prolyl hydroxylation-mediated suppression of RIPK1 to promote necroptosis and inflammation
Nature Cell Biology·2023·59 cit.·DOI
8
Ulk1-dependent alternative mitophagy plays a protective role during pressure overload in the heart
Cardiovascular Research·2022·57 cit.·DOI
9
Hepatic CEACAM1 expression indicates donor liver quality and prevents early transplantation injury
Journal of Clinical Investigation·2020·55 cit.·DOI
10
Ca2+-mediated mitochondrial inner membrane permeabilization induces cell death independently of Bax and Bak
Cell Death and Differentiation·2022·54 cit.·DOI

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