Cell Signaling Technology

Phospho-SMAD1 (Ser463/465)/ SMAD5 (Ser463/465)/ SMAD9 (Ser465/467) (D5B10) Rabbit Monoclonal Antibody

#13820 · $169

100
Citing papers
8.9k
Combined citations
Supplier page Find this product

Research areas

Cancer Cells and Metastasis30TGF-β signaling in diseases11Epigenetics and DNA Methylation9MicroRNA in disease regulation9Hedgehog Signaling Pathway Studies6Cytokine Signaling Pathways and Interactions6Autophagy in Disease and Therapy6Wnt/β-catenin signaling in development and cancer6

Papers citing this product

1
CXCR1 blockade selectively targets human breast cancer stem cells in vitro and in xenografts
Journal of Clinical Investigation·2010·738 cit.·DOI
2
Elimination of Microglia Improves Functional Outcomes Following Extensive Neuronal Loss in the Hippocampus
Journal of Neuroscience·2015·292 cit.·DOI
3
Isoliquiritin ameliorates depression by suppressing NLRP3-mediated pyroptosis via miRNA-27a/SYK/NF-κB axis
Journal of Neuroinflammation·2021·288 cit.·DOI
4
Characterization of LINE-1 Ribonucleoprotein Particles
PLoS Genetics·2010·245 cit.·DOI
5
Isolation and Characterization of Tumor Cells from the Ascites of Ovarian Cancer Patients: Molecular Phenotype of Chemoresistant Ovarian Tumors
PLoS ONE·2012·238 cit.·DOI
6
Inhibition of iNOS as a novel effective targeted therapy against triple-negative breast cancer
Breast Cancer Research·2015·221 cit.·DOI
7
Chemokine CCL2–CCR2 Signaling Induces Neuronal Cell Death via STAT3 Activation and IL-1β Production after Status Epilepticus
Journal of Neuroscience·2017·214 cit.·DOI
8
Leptin-induced Epithelial-Mesenchymal Transition in Breast Cancer Cells Requires β-Catenin Activation via Akt/GSK3- and MTA1/Wnt1 Protein-dependent Pathways
Journal of Biological Chemistry·2012·194 cit.·DOI
9
Blocking interleukin-6 trans-signaling protects against renal fibrosis by suppressing STAT3 activation
Theranostics·2019·184 cit.·DOI
10
LGR5 Promotes Breast Cancer Progression and Maintains Stem-Like Cells Through Activation of Wnt/β-Catenin Signaling
Stem Cells·2015·169 cit.·DOI

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