Cell Signaling Technology

Sall4 (D16H12) Rabbit Monoclonal Antibody

#8459 · $179

100
Citing papers
11.6k
Combined citations
Supplier page Find this product

Research areas

Wnt/β-catenin signaling in development and cancer27interferon and immune responses18Immune Response and Inflammation11Cancer-related gene regulation10NF-κB Signaling Pathways9Genetics and Neurodevelopmental Disorders9Endoplasmic Reticulum Stress and Disease7Autophagy in Disease and Therapy6

Papers citing this product

1
Axin-mediated CKI phosphorylation of β-catenin at Ser 45: a molecular switch for the Wnt pathway
Genes & Development·2002·771 cit.·DOI
2
TLR2 senses the SARS-CoV-2 envelope protein to produce inflammatory cytokines
Nature Immunology·2021·607 cit.·DOI
3
Microtubules Facilitate Autophagosome Formation and Fusion of Autophagosomes with Endosomes
Traffic·2005·420 cit.·DOI
4
In Vivo Regulation of Glycogen Synthase Kinase-3β (GSK3β) by Serotonergic Activity in Mouse Brain
Neuropsychopharmacology·2004·300 cit.·DOI
5
The Coxsackievirus B 3Cpro Protease Cleaves MAVS and TRIF to Attenuate Host Type I Interferon and Apoptotic Signaling
PLoS Pathogens·2011·299 cit.·DOI
6
Neuroprotective Effects of Regulators of the Glycogen Synthase Kinase-3β Signaling Pathway in a Transgenic Model of Alzheimer's Disease Are Associated with Reduced Amyloid Precursor Protein Phosphorylation
Journal of Neuroscience·2007·287 cit.·DOI
7
Attenuated mTOR Signaling and Enhanced Autophagy in Adipocytes from Obese Patients with Type 2 Diabetes
Molecular Medicine·2010·272 cit.·DOI
8
SLAB51 Probiotic Formulation Activates SIRT1 Pathway Promoting Antioxidant and Neuroprotective Effects in an AD Mouse Model
Molecular Neurobiology·2018·262 cit.·DOI
9
Full Reversal of Alzheimer's Disease-Like Phenotype in a Mouse Model with Conditional Overexpression of Glycogen Synthase Kinase-3
Journal of Neuroscience·2006·253 cit.·DOI
10
Lipopolysaccharide-induced interleukin-6 production is controlled by glycogen synthase kinase-3 and STAT3 in the brain
Journal of Neuroinflammation·2009·235 cit.·DOI

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