Cell Signaling Technology

PhosphoPlus® SAPK/JNK (Thr183/Tyr185) Antibody Duet

#8206 · $673

100
Citing papers
4.3k
Combined citations
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Research areas

MicroRNA in disease regulation9Muscle Physiology and Disorders9Neuroinflammation and Neurodegeneration Mechanisms7Extracellular vesicles in disease7Neuroscience and Neuropharmacology Research7Ubiquitin and proteasome pathways6Autophagy in Disease and Therapy6Neurogenesis and neuroplasticity mechanisms5

Papers citing this product

1
Antisense oligonucleotides increase <i>Scn1a</i> expression and reduce seizures and SUDEP incidence in a mouse model of Dravet syndrome
Science Translational Medicine·2020·332 cit.·DOI
2
Small, Nonpeptide p75<sup>NTR</sup>Ligands Induce Survival Signaling and Inhibit proNGF-Induced Death
Journal of Neuroscience·2006·178 cit.·DOI
3
The tetraspanin web revisited by super-resolution microscopy
Scientific Reports·2015·162 cit.·DOI
4
Proteomic Identification and Functional Validation of Activins and Bone Morphogenetic Protein 11 as Candidate Novel Muscle Mass Regulators
Molecular Endocrinology·2008·136 cit.·DOI
5
Two-color STED microscopy reveals different degrees of colocalization between hexokinase-I and the three human VDAC isoforms
PMC Biophysics·2010·132 cit.·DOI
6
H3K9me3 Inhibition Improves Memory, Promotes Spine Formation, and Increases BDNF Levels in the Aged Hippocampus
Journal of Neuroscience·2016·125 cit.·DOI
7
Targeted Ablation of Oligodendrocytes Induces Axonal Pathology Independent of Overt Demyelination
Journal of Neuroscience·2012·117 cit.·DOI
8
MiR-206 inhibits HGF-induced epithelial-mesenchymal transition and angiogenesis in non-small cell lung cancer via c-Met /PI3k/Akt/mTOR pathway
Oncotarget·2016·116 cit.·DOI
9
αV-class integrins exert dual roles on α5β1 integrins to strengthen adhesion to fibronectin
Nature Communications·2017·112 cit.·DOI
10
Ube2v1-mediated ubiquitination and degradation of Sirt1 promotes metastasis of colorectal cancer by epigenetically suppressing autophagy
Journal of Hematology & Oncology·2018·110 cit.·DOI

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