Cell Signaling Technology

Phospho-Acetyl-CoA Carboxylase (Ser79) Antibody

#3661 · $362,852

67
Evidence Score · Well-Established
Used by 197 independent labs across 209 papers. How we score this →
100
Citing papers
15.4k
Combined citations
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Research areas

Metabolism, Diabetes, and Cancer21Adipose Tissue and Metabolism15Autophagy in Disease and Therapy12Pancreatic function and diabetes11Adipokines, Inflammation, and Metabolic Diseases7Cancer, Hypoxia, and Metabolism7Cancer-related Molecular Pathways5Diet and metabolism studies5

Papers citing this product

1
Transcriptional control of autophagy–lysosome function drives pancreatic cancer metabolism
Nature·2015·788 cit.·DOI
2
AC220 is a uniquely potent and selective inhibitor of FLT3 for the treatment of acute myeloid leukemia (AML)
Blood·2009·641 cit.·DOI
3
Hexokinase-2 depletion inhibits glycolysis and induces oxidative phosphorylation in hepatocellular carcinoma and sensitizes to metformin
Nature Communications·2018·552 cit.·DOI
4
Glutathione depletion induces ferroptosis, autophagy, and premature cell senescence in retinal pigment epithelial cells
Cell Death and Disease·2018·537 cit.·DOI
5
Rev-erb-α modulates skeletal muscle oxidative capacity by regulating mitochondrial biogenesis and autophagy
Nature Medicine·2013·437 cit.·DOI
6
Metformin reduces liver glucose production by inhibition of fructose-1-6-bisphosphatase
Nature Medicine·2018·324 cit.·DOI
7
SIRT4 Regulates Fatty Acid Oxidation and Mitochondrial Gene Expression in Liver and Muscle Cells
Journal of Biological Chemistry·2010·321 cit.·DOI
8
Metformin alleviates hepatosteatosis by restoring SIRT1-mediated autophagy induction via an AMP-activated protein kinase-independent pathway
Autophagy·2014·319 cit.·DOI
9
The Coxsackievirus B 3Cpro Protease Cleaves MAVS and TRIF to Attenuate Host Type I Interferon and Apoptotic Signaling
PLoS Pathogens·2011·299 cit.·DOI
10
<scp>AMPK</scp> activation protects cells from oxidative stress‐induced senescence via autophagic flux restoration and intracellular <scp>NAD</scp> <sup>+</sup> elevation
Aging Cell·2016·297 cit.·DOI

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