Cell Signaling Technology

CHOP (D46F1) Rabbit Monoclonal Antibody

#5554 · $149

100
Citing papers
7.9k
Combined citations
Supplier page Find this product

Research areas

Endoplasmic Reticulum Stress and Disease13Cancer, Hypoxia, and Metabolism12Autophagy in Disease and Therapy10Chemokine receptors and signaling7NF-κB Signaling Pathways5Acute Myeloid Leukemia Research5Metabolism, Diabetes, and Cancer5T-cell and B-cell Immunology5

Papers citing this product

1
The eIF2α/ATF4 pathway is essential for stress-induced autophagy gene expression
Nucleic Acids Research·2013·1.1k cit.·DOI
2
Stromal Cell–Derived Factor-1α Confers Protection Against Myocardial Ischemia/Reperfusion Injury
Circulation·2007·314 cit.·DOI
3
Modulation of PKM alternative splicing by PTBP1 promotes gemcitabine resistance in pancreatic cancer cells
Oncogene·2015·283 cit.·DOI
4
Tumor Necrosis Factor (TNF) Receptor Shedding Controls Thresholds of Innate Immune Activation That Balance Opposing TNF Functions in Infectious and Inflammatory Diseases
The Journal of Experimental Medicine·2004·197 cit.·DOI
5
Thiostrepton induces ferroptosis in pancreatic cancer cells through STAT3/GPX4 signalling
Cell Death and Disease·2022·175 cit.·DOI
6
Immune Evasion by Murine Melanoma Mediated through CC Chemokine Receptor-10
The Journal of Experimental Medicine·2003·168 cit.·DOI
7
Promotion of epithelial-mesenchymal transformation by hepatocellular carcinoma-educated macrophages through Wnt2b/β-catenin/c-Myc signaling and reprogramming glycolysis
Journal of Experimental & Clinical Cancer Research·2021·160 cit.·DOI
8
Metabolic Reprogramming towards Aerobic Glycolysis Correlates with Greater Proliferative Ability and Resistance to Metabolic Inhibition in CD8 versus CD4 T Cells
PLoS ONE·2014·153 cit.·DOI
9
Shikonin Inhibits Tumor Growth in Mice by Suppressing Pyruvate Kinase M2-mediated Aerobic Glycolysis
Scientific Reports·2018·152 cit.·DOI
10
Platelet-Derived Growth Factor-D Overexpression Contributes to Epithelial-Mesenchymal Transition of PC3 Prostate Cancer Cells
Stem Cells·2008·151 cit.·DOI

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