Cell Signaling Technology

Phospho-GSK-3 beta (Ser9) (D85E12) Rabbit Monoclonal Antibody

#5558 · $179

100
Citing papers
12.6k
Combined citations
Supplier page Find this product

Research areas

PI3K/AKT/mTOR signaling in cancer12Neuroscience and Neuropharmacology Research9Tuberous Sclerosis Complex Research8Cancer, Hypoxia, and Metabolism6Neurogenesis and neuroplasticity mechanisms6Cancer Cells and Metastasis6Epigenetics and DNA Methylation6Hippo pathway signaling and YAP/TAZ5

Papers citing this product

1
Response of a Neuronal Model of Tuberous Sclerosis to Mammalian Target of Rapamycin (mTOR) Inhibitors: Effects on mTORC1 and Akt Signaling Lead to Improved Survival and Function
Journal of Neuroscience·2008·502 cit.·DOI
2
A Mouse Model of Tuberous Sclerosis: Neuronal Loss of Tsc1 Causes Dysplastic and Ectopic Neurons, Reduced Myelination, Seizure Activity, and Limited Survival
Journal of Neuroscience·2007·460 cit.·DOI
3
Pharmacological Inhibition of mTORC1 Suppresses Anatomical, Cellular, and Behavioral Abnormalities in Neural-Specific<i>Pten</i>Knock-Out Mice
Journal of Neuroscience·2009·439 cit.·DOI
4
Akt Signals through the Mammalian Target of Rapamycin Pathway to Regulate CNS Myelination
Journal of Neuroscience·2009·321 cit.·DOI
5
Ink4a-Arf loss cooperates with KRas activation in astrocytes and neural progenitors to generate glioblastomas of various morphologies depending on activated Akt.
PubMed·2002·292 cit.
6
Brain Cancer Stem Cells Display Preferential Sensitivity to Akt Inhibition
Stem Cells·2008·241 cit.·DOI
7
Loss of Tsc2 in radial glia models the brain pathology of tuberous sclerosis complex in the mouse
Human Molecular Genetics·2009·221 cit.·DOI
8
A TREM2-activating antibody with a blood–brain barrier transport vehicle enhances microglial metabolism in Alzheimer’s disease models
Nature Neuroscience·2023·217 cit.·DOI
9
FANCJ coordinates two pathways that maintain epigenetic stability at G-quadruplex DNA
Nucleic Acids Research·2011·215 cit.·DOI
10
Loss of Tsc1 In Vivo Impairs Hippocampal mGluR-LTD and Increases Excitatory Synaptic Function
Journal of Neuroscience·2011·204 cit.·DOI

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