Cell Signaling Technology

Phospho-PDGF Receptor beta (Tyr751) (88H8) Mouse Monoclonal Antibody

#3166 · western blot · $203,482

64
Evidence Score · Well-Established
Used by 288 independent labs across 302 papers. How we score this →
100
Citing papers
11.4k
Combined citations
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Research areas

Autophagy in Disease and Therapy8Cytokine Signaling Pathways and Interactions6Epigenetics and DNA Methylation5Neuroinflammation and Neurodegeneration Mechanisms5PARP inhibition in cancer therapy5Acute Myeloid Leukemia Research4DNA Repair Mechanisms4CRISPR and Genetic Engineering4

Papers citing this product

1
Comparative host-coronavirus protein interaction networks reveal pan-viral disease mechanisms
Science·2020·813 cit.·DOI
2
<i>FBW7</i> mutations in leukemic cells mediate NOTCH pathway activation and resistance to γ-secretase inhibitors
The Journal of Experimental Medicine·2007·660 cit.·DOI
3
Replication gaps are a key determinant of PARP inhibitor synthetic lethality with BRCA deficiency
Molecular Cell·2021·315 cit.·DOI
4
Synthetic lethality between androgen receptor signalling and the PARP pathway in prostate cancer
Nature Communications·2017·278 cit.·DOI
5
Effects of Copper Chelation on BRAFV600E Positive Colon Carcinoma Cells
Cancers·2019·212 cit.·DOI
6
Loss of Apelin Exacerbates Myocardial Infarction Adverse Remodeling and Ischemia‐reperfusion Injury: Therapeutic Potential of Synthetic Apelin Analogues
Journal of the American Heart Association·2013·211 cit.·DOI
7
Notch1 is an effector of Akt and hypoxia in melanoma development
Journal of Clinical Investigation·2008·207 cit.·DOI
8
ATP-binding Cassette Transporter A1 Expression Disrupts Raft Membrane Microdomains through Its ATPase-related Functions
Journal of Biological Chemistry·2006·207 cit.·DOI
9
Genistein inhibits radiation-induced activation of NF-κB in prostate cancer cells promoting apoptosis and G2/M cell cycle arrest
BMC Cancer·2006·201 cit.·DOI
10
PEGylated graphene oxide elicits strong immunological responses despite surface passivation
Nature Communications·2017·199 cit.·DOI

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