Cell Signaling Technology

Phospho-HER3/ErbB3 (Tyr1289) (D1B5) Rabbit Monoclonal Antibody (BSA and Azide Free)

#46661 · $898

16
Citing papers
635
Combined citations
Supplier page Find this product

Research areas

Platelet Disorders and Treatments4TGF-β signaling in diseases4Cancer-related gene regulation2Bone health and treatments2Antiplatelet Therapy and Cardiovascular Diseases2Pancreatic and Hepatic Oncology Research2Cancer Cells and Metastasis1Bone Metabolism and Diseases1

Papers citing this product

1
EGF induces epithelial-mesenchymal transition through phospho-Smad2/3-Snail signaling pathway in breast cancer cells
Oncotarget·2016·129 cit.·DOI
2
MicroRNA-182 targets SMAD7 to potentiate TGFβ-induced epithelial-mesenchymal transition and metastasis of cancer cells
Nature Communications·2016·126 cit.·DOI
3
Upregulation of Ets1 expression by NFATc2 and NFKB1/RELA promotes breast cancer cell invasiveness
Oncogenesis·2018·68 cit.·DOI
4
Amphiregulin-EGFR signaling regulates PTHrP gene expression in breast cancer cells
Breast Cancer Research and Treatment·2007·56 cit.·DOI
5
Differential Involvement of ERK2 and p38 in Platelet Adhesion to Collagen
Journal of Biological Chemistry·2005·55 cit.·DOI
6
Akt and mitogen-activated protein kinase enhance C-type lectin-like receptor 2-mediated platelet activation by inhibition of glycogen synthase kinase 3α/β
Journal of Thrombosis and Haemostasis·2015·46 cit.·DOI
7
Anti-platelet activity of panaxatriol saponins is mediated by suppression of intracellular calcium mobilization and ERK2/p38 activation
BMC Complementary and Alternative Medicine·2016·44 cit.·DOI
8
Ets-1 deficiency alleviates nonalcoholic steatohepatitis via weakening TGF-β1 signaling-mediated hepatocyte apoptosis
Cell Death and Disease·2019·33 cit.·DOI
9
15-Deoxy-Δ12,14-Prostaglandin J2 Inhibits Osteolytic Breast Cancer Bone Metastasis and Estrogen Deficiency-Induced Bone Loss
PLoS ONE·2015·29 cit.·DOI
10
Cyclic AMP Enhances TGFβ Responses of Breast Cancer Cells by Upregulating TGFβ Receptor I Expression
PLoS ONE·2013·21 cit.·DOI

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