Cell Signaling Technology

Ubiquityl-Histone H2B (Lys120) (D11) Rabbit Monoclonal Antibody

#5546 · $179

100
Citing papers
12.4k
Combined citations
Supplier page Find this product

Research areas

PI3K/AKT/mTOR signaling in cancer12Neuroscience and Neuropharmacology Research8Ubiquitin and proteasome pathways7Cell death mechanisms and regulation7Tuberous Sclerosis Complex Research7Cancer, Hypoxia, and Metabolism7Immunotherapy and Immune Responses7Chemokine receptors and signaling6

Papers citing this product

1
Response of a Neuronal Model of Tuberous Sclerosis to Mammalian Target of Rapamycin (mTOR) Inhibitors: Effects on mTORC1 and Akt Signaling Lead to Improved Survival and Function
Journal of Neuroscience·2008·502 cit.·DOI
2
A Mouse Model of Tuberous Sclerosis: Neuronal Loss of Tsc1 Causes Dysplastic and Ectopic Neurons, Reduced Myelination, Seizure Activity, and Limited Survival
Journal of Neuroscience·2007·460 cit.·DOI
3
Pharmacological Inhibition of mTORC1 Suppresses Anatomical, Cellular, and Behavioral Abnormalities in Neural-Specific<i>Pten</i>Knock-Out Mice
Journal of Neuroscience·2009·439 cit.·DOI
4
The experimental power of FR900359 to study Gq-regulated biological processes
Nature Communications·2015·371 cit.·DOI
5
Akt Signals through the Mammalian Target of Rapamycin Pathway to Regulate CNS Myelination
Journal of Neuroscience·2009·321 cit.·DOI
6
Stromal Cell–Derived Factor-1α Confers Protection Against Myocardial Ischemia/Reperfusion Injury
Circulation·2007·314 cit.·DOI
7
Fluid shear stress activates YAP1 to promote cancer cell motility
Nature Communications·2017·259 cit.·DOI
8
Targeting the CoREST complex with dual histone deacetylase and demethylase inhibitors
Nature Communications·2018·231 cit.·DOI
9
Non-small cell lung cancer cells survived ionizing radiation treatment display cancer stem cell and epithelial-mesenchymal transition phenotypes
Molecular Cancer·2013·226 cit.·DOI
10
Loss of Tsc2 in radial glia models the brain pathology of tuberous sclerosis complex in the mouse
Human Molecular Genetics·2009·221 cit.·DOI

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