Cell Signaling Technology

CHAF1A (D77D5) Rabbit Monoclonal Antibody

#5480 · $175

100
Citing papers
10.1k
Combined citations
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Research areas

Cell death mechanisms and regulation14Inflammasome and immune disorders11interferon and immune responses8Immune Response and Inflammation8NF-κB Signaling Pathways7Immune cells in cancer7CRISPR and Genetic Engineering6Epigenetics and DNA Methylation5

Papers citing this product

1
RIP1 autophosphorylation is promoted by mitochondrial ROS and is essential for RIP3 recruitment into necrosome
Nature Communications·2017·596 cit.·DOI
2
RIPK3 deficiency or catalytically inactive RIPK1 provides greater benefit than MLKL deficiency in mouse models of inflammation and tissue injury
Cell Death and Differentiation·2016·439 cit.·DOI
3
Hemangioblastic Derivatives from Human Induced Pluripotent Stem Cells Exhibit Limited Expansion and Early Senescence
Stem Cells·2010·374 cit.·DOI
4
Regulation of RIPK1 activation by TAK1-mediated phosphorylation dictates apoptosis and necroptosis
Nature Communications·2017·326 cit.·DOI
5
Cancer Stem Cells Contribute to Cisplatin Resistance in <i>Brca1/p53</i> –Mediated Mouse Mammary Tumors
Cancer Research·2008·302 cit.·DOI
6
Novel type II anti-CD20 monoclonal antibody (GA101) evokes homotypic adhesion and actin-dependent, lysosome-mediated cell death in B-cell malignancies
Blood·2011·290 cit.·DOI
7
The SARS Coronavirus 3a Protein Causes Endoplasmic Reticulum Stress and Induces Ligand-Independent Downregulation of the Type 1 Interferon Receptor
PLoS ONE·2009·271 cit.·DOI
8
TAK1 restricts spontaneous NLRP3 activation and cell death to control myeloid proliferation
The Journal of Experimental Medicine·2018·254 cit.·DOI
9
Autophosphorylation at serine 166 regulates RIP kinase 1-mediated cell death and inflammation
Nature Communications·2020·234 cit.·DOI
10
ADAR1 mutation causes ZBP1-dependent immunopathology
Nature·2022·207 cit.·DOI
CHAF1A (D77D5) Rabbit Monoclonal Antibody — Evidence — PubIndex