Cell Signaling Technology

HSPA8 (D12F2) Rabbit Monoclonal Antibody

#8444 · $161,351

100
Citing papers
3k
Combined citations
Supplier page Find this product

Research areas

Nuclear Receptors and Signaling10Wnt/β-catenin signaling in development and cancer9DNA Repair Mechanisms8Immune Cell Function and Interaction8Immune cells in cancer7RNA Research and Splicing6Macrophage Migration Inhibitory Factor6RNA modifications and cancer6

Papers citing this product

1
Lipopolysaccharide-induced interleukin-6 production is controlled by glycogen synthase kinase-3 and STAT3 in the brain
Journal of Neuroinflammation·2009·235 cit.·DOI
2
DNAM-1 Expression Marks an Alternative Program of NK Cell Maturation
Cell Reports·2015·125 cit.·DOI
3
Glycogen synthase kinase-3: a new therapeutic target in renal cell carcinoma
British Journal of Cancer·2009·107 cit.·DOI
4
Factor XIIIA—expressing inflammatory monocytes promote lung squamous cancer through fibrin cross-linking
Nature Communications·2018·104 cit.·DOI
5
1,1-Bis(3′-indolyl)-1-( <i>p</i> -chlorophenyl)methane activates the orphan nuclear receptor Nurr1 and inhibits bladder cancer growth
Molecular Cancer Therapeutics·2008·104 cit.·DOI
6
Reactive Oxygen Species– and DNA Damage Response–Dependent NK Cell Activating Ligand Upregulation Occurs at Transcriptional Levels and Requires the Transcriptional Factor E2F1
The Journal of Immunology·2014·98 cit.·DOI
7
Hepatocyte Growth Factor Suppresses Proinflammatory NFκB Activation through GSK3β Inactivation in Renal Tubular Epithelial Cells
Journal of Biological Chemistry·2008·97 cit.·DOI
8
The Involvement of MAPK Signaling Pathways in Determining the Cellular Response to p53 Activation
Journal of Biological Chemistry·2005·88 cit.·DOI
9
Targeting miR-381-NEFL axis sensitizes glioblastoma cells to temozolomide by regulating stemness factors and multidrug resistance factors
Oncotarget·2014·84 cit.·DOI
10
KPNB1 inhibition disrupts proteostasis and triggers unfolded protein response-mediated apoptosis in glioblastoma cells
Oncogene·2018·73 cit.·DOI

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