Cell Signaling Technology

COX IV Antibody

#4844 · $314

100
Citing papers
10.9k
Combined citations
Supplier page Find this product

Research areas

Mitochondrial Function and Pathology14Adipose Tissue and Metabolism11Autophagy in Disease and Therapy9Peroxisome Proliferator-Activated Receptors8Cell death mechanisms and regulation8Alzheimer's disease research and treatments6Muscle Physiology and Disorders6Immune cells in cancer5

Papers citing this product

1
Brown adipose tissue regulates glucose homeostasis and insulin sensitivity
Journal of Clinical Investigation·2012·1.2k cit.·DOI
2
Isocitrate dehydrogenase mutations suppress STAT1 and CD8+ T cell accumulation in gliomas
Journal of Clinical Investigation·2017·411 cit.·DOI
3
Requirement for the histone deacetylase Hdac3 for the inflammatory gene expression program in macrophages
Proceedings of the National Academy of Sciences·2012·395 cit.·DOI
4
Transforming Growth Factor-β-induced Apoptosis Is Mediated by Smad-dependent Expression of GADD45b through p38 Activation
Journal of Biological Chemistry·2003·256 cit.·DOI
5
ATF3/SPI1/SLC31A1 Signaling Promotes Cuproptosis Induced by Advanced Glycosylation End Products in Diabetic Myocardial Injury
International Journal of Molecular Sciences·2023·204 cit.·DOI
6
Activation of mTORC1 is essential for β-adrenergic stimulation of adipose browning
Journal of Clinical Investigation·2016·196 cit.·DOI
7
Moderate-Intensity Exercise and High-Intensity Interval Training Affect Insulin Sensitivity Similarly in Obese Adults
The Journal of Clinical Endocrinology & Metabolism·2020·195 cit.·DOI
8
PPAR-γ Ligands Repress TGFβ-Induced Myofibroblast Differentiation by Targeting the PI3K/Akt Pathway: Implications for Therapy of Fibrosis
PLoS ONE·2011·189 cit.·DOI
9
CKAP4 is a Dickkopf1 receptor and is involved in tumor progression
Journal of Clinical Investigation·2016·182 cit.·DOI
10
Peroxisome Proliferator-Activated Receptor-γ Modulates the Response of Macrophages to Lipopolysaccharide and Glucocorticoids
Frontiers in Immunology·2018·165 cit.·DOI

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