Abnova

SQSTM1 monoclonal antibody (M01), clone 2C11

#H00008878-M01 · western blot

60
Evidence Score · Well-Established
Used by 51 independent labs across 148 papers. How we score this →
148
Citing papers
5.6k
Combined citations
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Research areas

Autophagy in Disease and Therapy43Endoplasmic Reticulum Stress and Disease12Ubiquitin and proteasome pathways7Alzheimer's disease research and treatments6Lipid metabolism and biosynthesis5Cellular transport and secretion5Lysosomal Storage Disorders Research4Advanced Glycation End Products research4

Papers citing this product

1
Autosis is a Na <sup>+</sup> ,K <sup>+</sup> -ATPase–regulated form of cell death triggered by autophagy-inducing peptides, starvation, and hypoxia–ischemia
Proceedings of the National Academy of Sciences·2013·577 cit.·DOI
2
C9orf72 frontotemporal lobar degeneration is characterised by frequent neuronal sense and antisense RNA foci
Acta Neuropathologica·2013·332 cit.·DOI
3
A selective <scp>ER</scp> ‐phagy exerts procollagen quality control via a Calnexin‐ <scp>FAM</scp> 134B complex
The EMBO Journal·2018·253 cit.·DOI
4
Defective autophagy in neurons and astrocytes from mice deficient in PI(3,5)P2
Human Molecular Genetics·2009·203 cit.·DOI
5
Regulation of YAP by mTOR and autophagy reveals a therapeutic target of tuberous sclerosis complex
The Journal of Experimental Medicine·2014·198 cit.·DOI
6
Critical Role of FoxO3a in Alcohol-Induced Autophagy and Hepatotoxicity
American Journal Of Pathology·2013·164 cit.·DOI
7
A Becn1 mutation mediates hyperactive autophagic sequestration of amyloid oligomers and improved cognition in Alzheimer's disease
PLoS Genetics·2017·160 cit.·DOI
8
Upregulation of Rubicon promotes autosis during myocardial ischemia/reperfusion injury
Journal of Clinical Investigation·2020·146 cit.·DOI
9
The flavonoid 4,4′-dimethoxychalcone promotes autophagy-dependent longevity across species
Nature Communications·2019·140 cit.·DOI
10
Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2–JNK signalling in breast cancer
Nature Communications·2016·134 cit.·DOI

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