Cell Signaling Technology

SMAD5 (D4G2) Rabbit Monoclonal Antibody

#12534 · western blot · $157,348

65
Evidence Score · Well-Established
Used by 196 independent labs across 204 papers. How we score this →
100
Citing papers
3.9k
Combined citations
Supplier page Find this product

Research areas

Inflammasome and immune disorders21Autophagy in Disease and Therapy12Mitochondrial Function and Pathology8interferon and immune responses6Endoplasmic Reticulum Stress and Disease6Cell death mechanisms and regulation6Circadian rhythm and melatonin5RNA modifications and cancer5

Papers citing this product

1
Genome-wide CRISPR screen identifies host dependency factors for influenza A virus infection
Nature Communications·2020·244 cit.·DOI
2
MFN2 suppresses cancer progression through inhibition of mTORC2/Akt signaling
Scientific Reports·2017·159 cit.·DOI
3
Actionable perturbations of damage responses by TCL1/ATM and epigenetic lesions form the basis of T-PLL
Nature Communications·2018·105 cit.·DOI
4
Selective inhibition of the p38α MAPK–MK2 axis inhibits inflammatory cues including inflammasome priming signals
The Journal of Experimental Medicine·2018·103 cit.·DOI
5
M6A associated TSUC7 inhibition contributed to Erlotinib resistance in lung adenocarcinoma through a notch signaling activation dependent way
Journal of Experimental & Clinical Cancer Research·2021·92 cit.·DOI
6
A Therapeutic Peptide Vaccine Against PCSK9
Scientific Reports·2017·89 cit.·DOI
7
A novel IFNα-induced long noncoding RNA negatively regulates immunosuppression by interrupting H3K27 acetylation in head and neck squamous cell carcinoma
Molecular Cancer·2020·88 cit.·DOI
8
Blockade of Autophagy Prevents the Progression of Hyperuricemic Nephropathy Through Inhibiting NLRP3 Inflammasome-Mediated Pyroptosis
Frontiers in Immunology·2022·87 cit.·DOI
9
Lactylation stabilizes TFEB to elevate autophagy and lysosomal activity
The Journal of Cell Biology·2024·86 cit.·DOI
10
TMBIM6/BI-1 contributes to cancer progression through assembly with mTORC2 and AKT activation
Nature Communications·2020·80 cit.·DOI

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