Cell Signaling Technology

Phospho-PDGF Receptor alpha (Tyr1018) Antibody

#4547 · western blot · $148,362

100
Citing papers
10.5k
Combined citations
Supplier page Find this product

Research areas

Neurogenesis and neuroplasticity mechanisms16Neuroinflammation and Neurodegeneration Mechanisms13Nerve injury and regeneration9NF-κB Signaling Pathways6Immune Response and Inflammation6MicroRNA in disease regulation6Neuroscience and Neuropharmacology Research6Adipose Tissue and Metabolism5

Papers citing this product

1
Mutational signatures in esophageal adenocarcinoma define etiologically distinct subgroups with therapeutic relevance
Nature Genetics·2016·433 cit.·DOI
2
Hypoxia and defective apoptosis drive genomic instability and tumorigenesis
Genes & Development·2004·277 cit.·DOI
3
Early glycolytic reprogramming controls microglial inflammatory activation
Journal of Neuroinflammation·2021·254 cit.·DOI
4
Internalized Tau Oligomers Cause Neurodegeneration by Inducing Accumulation of Pathogenic Tau in Human Neurons Derived from Induced Pluripotent Stem Cells
Journal of Neuroscience·2015·252 cit.·DOI
5
Serotonin Is Required for Exercise-Induced Adult Hippocampal Neurogenesis
Journal of Neuroscience·2013·231 cit.·DOI
6
Retinoic Acid Induces Blood–Brain Barrier Development
Journal of Neuroscience·2013·204 cit.·DOI
7
Neutrophils Are Critical for Myelin Removal in a Peripheral Nerve Injury Model of Wallerian Degeneration
Journal of Neuroscience·2017·190 cit.·DOI
8
Epigallocatechin, a Green Tea Polyphenol, Attenuates Myocardial Ischemia Reperfusion Injury in Rats
Molecular Medicine·2004·181 cit.·DOI
9
A Novel Role of the WNT-Dishevelled-GSK3  Signaling Cascade in the Mouse Nucleus Accumbens in a Social Defeat Model of Depression
Journal of Neuroscience·2011·172 cit.·DOI
10
Hypercoagulability causes atrial fibrosis and promotes atrial fibrillation
European Heart Journal·2016·170 cit.·DOI

Rate this reagent

Used this in your lab? Leave a quick rating. Reviews are anonymous and verified by academic email before they appear.

Your email is never shown publicly.