Cell Signaling Technology

RUNX3/AML2 (D6E2) Rabbit Monoclonal Antibody (Alexa Fluor® 488 Conjugate)

#25330 · $465

21
Citing papers
801
Combined citations
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Research areas

Endoplasmic Reticulum Stress and Disease3Cancer therapeutics and mechanisms3Metabolism, Diabetes, and Cancer3Melanoma and MAPK Pathways2Cancer-related molecular mechanisms research2FOXO transcription factor regulation2Advanced biosensing and bioanalysis techniques2DNA and Nucleic Acid Chemistry2

Papers citing this product

1
c‐Jun N‐terminal kinase mediates mouse liver injury through a novel Sab (SH3BP5)‐dependent pathway leading to inactivation of intramitochondrial Src
Hepatology·2016·186 cit.·DOI
2
Insulin receptor substrates mediate distinct biological responses to insulin-like growth factor receptor activation in breast cancer cells
British Journal of Cancer·2006·118 cit.·DOI
3
Elevated MicroRNA-31 Expression Regulates Colorectal Cancer Progression by Repressing Its Target Gene SATB2
PLoS ONE·2013·105 cit.·DOI
4
N6-Methyladenosine RNA Demethylase FTO Promotes Gastric Cancer Metastasis by Down-Regulating the m6A Methylation of ITGB1
Frontiers in Oncology·2021·66 cit.·DOI
5
BAF60c prevents abdominal aortic aneurysm formation through epigenetic control of vascular smooth muscle cell homeostasis
Journal of Clinical Investigation·2022·60 cit.·DOI
6
Cytolethal Distending Toxins Require Components of the ER-Associated Degradation Pathway for Host Cell Entry
PLoS Pathogens·2014·49 cit.·DOI
7
Enterovirus 71 protease 2Apro and 3Cpro differentially inhibit the cellular endoplasmic reticulum-associated degradation (ERAD) pathway via distinct mechanisms, and enterovirus 71 hijacks ERAD component p97 to promote its replication
PLoS Pathogens·2017·43 cit.·DOI
8
Inhibition of ERAD synergizes with FTS to eradicate pancreatic cancer cells
BMC Cancer·2021·38 cit.·DOI
9
Negative Regulators of Insulin Signaling Revealed in a Genome-Wide Functional Screen
PLoS ONE·2009·36 cit.·DOI
10
ERK2-topoisomerase II regulatory axis is important for gene activation in immediate early genes
Nature Communications·2023·23 cit.·DOI

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