Cell Signaling Technology

GMPS Antibody

#14602 · $317

100
Citing papers
6.9k
Combined citations
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Research areas

Metabolism, Diabetes, and Cancer44Adipose Tissue and Metabolism22Pancreatic function and diabetes20Protein Kinase Regulation and GTPase Signaling12Muscle Physiology and Disorders10PI3K/AKT/mTOR signaling in cancer8Muscle metabolism and nutrition514-3-3 protein interactions5

Papers citing this product

1
Interleukin-1β-Induced Insulin Resistance in Adipocytes through Down-Regulation of Insulin Receptor Substrate-1 Expression
Endocrinology·2006·675 cit.·DOI
2
Distinct Signals Regulate AS160 Phosphorylation in Response to Insulin, AICAR, and Contraction in Mouse Skeletal Muscle
Diabetes·2006·316 cit.·DOI
3
Deletion of IFT20 in the mouse kidney causes misorientation of the mitotic spindle and cystic kidney disease
The Journal of Cell Biology·2008·290 cit.·DOI
4
Discovery of TBC1D1 as an Insulin-, AICAR-, and Contraction-stimulated Signaling Nexus in Mouse Skeletal Muscle
Journal of Biological Chemistry·2008·270 cit.·DOI
5
Organogenesis relies on SoxC transcription factors for the survival of neural and mesenchymal progenitors
Nature Communications·2010·229 cit.·DOI
6
Exercise training increases insulin-stimulated glucose disposal and GLUT4 (SLC2A4) protein content in patients with type 2 diabetes
Diabetologia·2006·224 cit.·DOI
7
Complementary regulation of TBC1D1 and AS160 by growth factors, insulin and AMPK activators
Biochemical Journal·2007·203 cit.·DOI
8
A Role for 14-3-3 in Insulin-stimulated GLUT4 Translocation through Its Interaction with the RabGAP AS160
Journal of Biological Chemistry·2006·200 cit.·DOI
9
Substrate specificity and effect on GLUT4 translocation of the Rab GTPase-activating protein Tbc1d1
Biochemical Journal·2007·194 cit.·DOI
10
Inhibition of GLUT4 Translocation by Tbc1d1, a Rab GTPase-activating Protein Abundant in Skeletal Muscle, Is Partially Relieved by AMP-activated Protein Kinase Activation
Journal of Biological Chemistry·2008·156 cit.·DOI