Cell Signaling Technology

Caspase-4 Antibody

#4450 · $314

100
Citing papers
8.3k
Combined citations
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Research areas

Autophagy in Disease and Therapy10Cell death mechanisms and regulation9Epigenetics and DNA Methylation7NF-κB Signaling Pathways6Endoplasmic Reticulum Stress and Disease5Immune Response and Inflammation5Inflammasome and immune disorders5Chronic Lymphocytic Leukemia Research5

Papers citing this product

1
The matrix component biglycan is proinflammatory and signals through Toll-like receptors 4 and 2 in macrophages
Journal of Clinical Investigation·2005·836 cit.·DOI
2
TNF signaling drives myeloid-derived suppressor cell accumulation
Journal of Clinical Investigation·2012·371 cit.·DOI
3
Mouse Retinal Cell Atlas: Molecular Identification of over Sixty Amacrine Cell Types
Journal of Neuroscience·2020·357 cit.·DOI
4
Ellagic acid, a phenolic compound, exerts anti-angiogenesis effects via VEGFR-2 signaling pathway in breast cancer
Breast Cancer Research and Treatment·2012·204 cit.·DOI
5
Loss of programmed cell death 4 expression marks adenoma‐carcinoma transition, correlates inversely with phosphorylated protein kinase B, and is an independent prognostic factor in resected colorectal cancer
Cancer·2007·197 cit.·DOI
6
Matrix metalloproteinase-9 inhibition ameliorates pathogenesis and improves skeletal muscle regeneration in muscular dystrophy
Human Molecular Genetics·2009·175 cit.·DOI
7
Sclerostin Inhibition of Wnt-3a-induced C3H10T1/2 Cell Differentiation Is Indirect and Mediated by Bone Morphogenetic Proteins
Journal of Biological Chemistry·2004·164 cit.·DOI
8
Complement pathway amplifies caspase-11–dependent cell death and endotoxin-induced sepsis severity
The Journal of Experimental Medicine·2016·151 cit.·DOI
9
Presenilin Is Necessary for Efficient Proteolysis through the Autophagy–Lysosome System in a γ-Secretase-Independent Manner
Journal of Neuroscience·2011·137 cit.·DOI
10
Cytochrome P450 2E1 (CYP2E1) regulates the response to oxidative stress and migration of breast cancer cells
Breast Cancer Research·2013·124 cit.·DOI

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