Cell Signaling Technology

AMFR Antibody

#9590 · $317

100
Citing papers
6.4k
Combined citations
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Research areas

Neuroscience and Neuropharmacology Research13Endoplasmic Reticulum Stress and Disease8Receptor Mechanisms and Signaling8Adipose Tissue and Metabolism6Ion channel regulation and function6Genomics, phytochemicals, and oxidative stress5Neuroinflammation and Neurodegeneration Mechanisms5Pancreatic function and diabetes5

Papers citing this product

1
Elevated sensitivity to diet-induced obesity and insulin resistance in mice lacking 4E-BP1 and 4E-BP2
Journal of Clinical Investigation·2007·332 cit.·DOI
2
Perilipin Promotes Hormone-sensitive Lipase-mediated Adipocyte Lipolysis via Phosphorylation-dependent and -independent Mechanisms
Journal of Biological Chemistry·2006·319 cit.·DOI
3
NLRP3/caspase-1/GSDMD–mediated pyroptosis exerts a crucial role in astrocyte pathological injury in mouse model of depression
JCI Insight·2021·308 cit.·DOI
4
Plant and Animal Pathogen Recognition Receptors Signal through Non-RD Kinases
PLoS Pathogens·2006·277 cit.·DOI
5
Regulation of Eukaryotic Initiation Factor 4E by Converging Signaling Pathways during Metabotropic Glutamate Receptor-Dependent Long-Term Depression
Journal of Neuroscience·2006·244 cit.·DOI
6
Inhibition of AMP-Activated Protein Kinase Signaling Alleviates Impairments in Hippocampal Synaptic Plasticity Induced by Amyloid β
Journal of Neuroscience·2014·195 cit.·DOI
7
Mitogen-Activated Protein Kinase Upregulates the Dendritic Translation Machinery in Long-Term Potentiation by Controlling the Mammalian Target of Rapamycin Pathway
Journal of Neuroscience·2007·170 cit.·DOI
8
Multiple Gq-Coupled Receptors Converge on a Common Protein Synthesis-Dependent Long-Term Depression That Is Affected in Fragile X Syndrome Mental Retardation
Journal of Neuroscience·2007·164 cit.·DOI
9
Removal of S6K1 and S6K2 leads to divergent alterations in learning, memory, and synaptic plasticity
Learning & Memory·2008·138 cit.·DOI
10
Hypoxia and TGF-β1 induced PLOD2 expression improve the migration and invasion of cervical cancer cells by promoting epithelial-to-mesenchymal transition (EMT) and focal adhesion formation
Cancer Cell International·2017·119 cit.·DOI