Cell Signaling Technology

Mitofusin-2 (D2D10) Rabbit Monoclonal Antibody

#9482 · $164

100
Citing papers
6.8k
Combined citations
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Research areas

Mitochondrial Function and Pathology12Autophagy in Disease and Therapy10Ubiquitin and proteasome pathways8Endoplasmic Reticulum Stress and Disease8Muscle Physiology and Disorders7MicroRNA in disease regulation6RNA modifications and cancer6Cell Adhesion Molecules Research5

Papers citing this product

1
Elicitation of Potent Neutralizing Antibody Responses by Designed Protein Nanoparticle Vaccines for SARS-CoV-2
Cell·2020·577 cit.·DOI
2
Global profiling of co- and post-translationally N-myristoylated proteomes in human cells
Nature Communications·2014·242 cit.·DOI
3
IL-6- and NGF-Induced Rapid Control of Protein Synthesis and Nociceptive Plasticity via Convergent Signaling to the eIF4F Complex
Journal of Neuroscience·2010·225 cit.·DOI
4
High-potency ligands for DREADD imaging and activation in rodents and monkeys
Nature Communications·2019·216 cit.·DOI
5
The mitochondria‐targeted anti‐oxidant MitoQ protects against intervertebral disc degeneration by ameliorating mitochondrial dysfunction and redox imbalance
Cell Proliferation·2020·198 cit.·DOI
6
AMP-activated Protein Kinase Activation Increases Phosphorylation of Glycogen Synthase Kinase 3β and Thereby Reduces cAMP-responsive Element Transcriptional Activity and Phosphoenolpyruvate Carboxykinase C Gene Expression in the Liver
Journal of Biological Chemistry·2008·189 cit.·DOI
7
Doxorubicin-induced cardiomyopathy associated with inhibition of autophagic degradation process and defects in mitochondrial respiration
Scientific Reports·2019·182 cit.·DOI
8
VEGF–integrin interplay controls tumor growth and vascularization
Proceedings of the National Academy of Sciences·2005·177 cit.·DOI
9
Desmoglein-1/Erbin interaction suppresses ERK activation to support epidermal differentiation
Journal of Clinical Investigation·2013·139 cit.·DOI
10
Role of Matrix Metalloproteinase-9 Dimers in Cell Migration
Journal of Biological Chemistry·2010·139 cit.·DOI

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