Cell Signaling Technology

Phospho-53BP1 (Ser25/29) Antibody

#2674 · $362

100
Citing papers
12.3k
Combined citations
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Research areas

Neuroinflammation and Neurodegeneration Mechanisms10Autophagy in Disease and Therapy8Cancer-related molecular mechanisms research6Immunotherapy and Immune Responses5DNA Repair Mechanisms5Immune Cell Function and Interaction5Alzheimer's disease research and treatments5Endoplasmic Reticulum Stress and Disease5

Papers citing this product

1
Isocitrate dehydrogenase mutations suppress STAT1 and CD8+ T cell accumulation in gliomas
Journal of Clinical Investigation·2017·411 cit.·DOI
2
Tumor Necrosis Factor-α, Interleukin-1β, and Interferon-γ Stimulate γ-Secretase-mediated Cleavage of Amyloid Precursor Protein through a JNK-dependent MAPK Pathway
Journal of Biological Chemistry·2004·384 cit.·DOI
3
Microfluidic Endothelium for Studying the Intravascular Adhesion of Metastatic Breast Cancer Cells
PLoS ONE·2009·311 cit.·DOI
4
Attenuated mTOR Signaling and Enhanced Autophagy in Adipocytes from Obese Patients with Type 2 Diabetes
Molecular Medicine·2010·272 cit.·DOI
5
IκB Kinase Regulates Social Defeat Stress-Induced Synaptic and Behavioral Plasticity
Journal of Neuroscience·2011·271 cit.·DOI
6
The chromatin-remodeling factor CHD4 coordinates signaling and repair after DNA damage
The Journal of Cell Biology·2010·248 cit.·DOI
7
Regulatory T cells trigger effector T cell DNA damage and senescence caused by metabolic competition
Nature Communications·2018·222 cit.·DOI
8
SOX2 regulates self-renewal and tumorigenicity of human melanoma-initiating cells
Oncogene·2014·202 cit.·DOI
9
The Cyclin-dependent Kinase Inhibitor CYC202 (<b><i>R</i></b>-Roscovitine) Inhibits Retinoblastoma Protein Phosphorylation, Causes Loss of Cyclin D1, and Activates the Mitogen-activated Protein Kinase Pathway
Cancer Research·2004·194 cit.·DOI
10
Prostate-specific membrane antigen cleavage of vitamin B9 stimulates oncogenic signaling through metabotropic glutamate receptors
The Journal of Experimental Medicine·2017·193 cit.·DOI