Cell Signaling Technology

Phospho-GRB10 (Ser476) (D4E6) Rabbit Monoclonal Antibody

#11817 · $176

100
Citing papers
3.3k
Combined citations
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Research areas

interferon and immune responses17Adipose Tissue and Metabolism6RNA modifications and cancer6NF-κB Signaling Pathways5Curcumin's Biomedical Applications5RNA Interference and Gene Delivery5Cytomegalovirus and herpesvirus research5RNA regulation and disease5

Papers citing this product

1
Phosphorylation of eucaryotic translation initiation factor 4B Ser422 is modulated by S6 kinases
The EMBO Journal·2004·461 cit.·DOI
2
Control of TANK-binding Kinase 1-mediated Signaling by the γ134.5 Protein of Herpes Simplex Virus 1
Journal of Biological Chemistry·2008·190 cit.·DOI
3
A DHX9-lncRNA-MDM2 interaction regulates cell invasion and angiogenesis of cervical cancer
Cell Death and Differentiation·2018·168 cit.·DOI
4
PLP2, a potent deubiquitinase from murine hepatitis virus, strongly inhibits cellular type I interferon production
Cell Research·2008·139 cit.·DOI
5
RIG-I-mediated Activation of p38 MAPK Is Essential for Viral Induction of Interferon and Activation of Dendritic Cells
Journal of Biological Chemistry·2009·119 cit.·DOI
6
TRAF Family Member-associated NF-κB Activator (TANK) Inhibits Genotoxic Nuclear Factor κB Activation by Facilitating Deubiquitinase USP10-dependent Deubiquitination of TRAF6 Ligase
Journal of Biological Chemistry·2015·107 cit.·DOI
7
Solitary fibrous tumors: loss of chimeric protein expression and genomic instability mark dedifferentiation
Modern Pathology·2015·95 cit.·DOI
8
A selective inhibitor of mitofusin 1-βIIPKC association improves heart failure outcome in rats
Nature Communications·2019·94 cit.·DOI
9
MicroRNA-31 negatively regulates peripherally derived regulatory T-cell generation by repressing retinoic acid-inducible protein 3
Nature Communications·2015·93 cit.·DOI
10
Caspase‐8 mutations in head and neck cancer confer resistance to death receptor‐mediated apoptosis and enhance migration, invasion, and tumor growth
Molecular Oncology·2014·84 cit.·DOI