Abcam

Synaptophysin antibody [SY38]

#ab8049 · western blot

151

Citing papers

5k

Combined citations

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Research areas

Alzheimer's disease research and treatments17 Neuroscience and Neuropharmacology Research14 Neuroinflammation and Neurodegeneration Mechanisms10 Parkinson's Disease Mechanisms and Treatments6 Amyotrophic Lateral Sclerosis Research5 Genetic Neurodegenerative Diseases4 Cholinesterase and Neurodegenerative Diseases4 Neurogenesis and neuroplasticity mechanisms4

Papers citing this product

1

Tau oligomers impair memory and induce synaptic and mitochondrial dysfunction in wild-type mice

Molecular Neurodegeneration·2011·614 cit.·DOI

2

The Synaptic Accumulation of Hyperphosphorylated Tau Oligomers in Alzheimer Disease Is Associated With Dysfunction of the Ubiquitin-Proteasome System

American Journal Of Pathology·2012·456 cit.·DOI

3

TDP-43 gains function due to perturbed autoregulation in a Tardbp knock-in mouse model of ALS-FTD

Nature Neuroscience·2018·294 cit.·DOI

4

Changes in Synaptic Proteins Precede Neurodegeneration Markers in Preclinical Alzheimer's Disease Cerebrospinal Fluid

Molecular & Cellular Proteomics·2019·252 cit.·DOI

5

Synaptic oligomeric tau in Alzheimer’s disease — A potential culprit in the spread of tau pathology through the brain

Neuron·2023·199 cit.·DOI

6

Hippocampal Function Is Impaired by a Short-Term High-Fat Diet in Mice: Increased Blood–Brain Barrier Permeability and Neuroinflammation as Triggering Events

Frontiers in Neuroscience·2021·157 cit.·DOI

7

Lipopolysaccharide-induced neuroinflammation induces presynaptic disruption through a direct action on brain tissue involving microglia-derived interleukin 1 beta

Journal of Neuroinflammation·2019·146 cit.·DOI

8

Frequent and symmetric deposition of misfolded tau oligomers within presynaptic and postsynaptic terminals in Alzheimer’s disease

Acta Neuropathologica Communications·2014·145 cit.·DOI

9

The neuritic plaque facilitates pathological conversion of tau in an Alzheimer’s disease mouse model

Nature Communications·2016·130 cit.·DOI

10

Loss of TREM2 rescues hyperactivation of microglia, but not lysosomal deficits and neurotoxicity in models of progranulin deficiency

The EMBO Journal·2022·92 cit.·DOI

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