Cell Signaling Technology

CD177 (F9O5U) Rabbit Monoclonal Antibody

#18991 · western blot · $178,415

63
Citing papers
2.2k
Combined citations
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Research areas

Glycosylation and Glycoproteins Research12Cell death mechanisms and regulation6Immunotherapy and Immune Responses5Cell Adhesion Molecules Research5Cancer Cells and Metastasis4Cancer Immunotherapy and Biomarkers4Galectins and Cancer Biology4Pancreatic and Hepatic Oncology Research4

Papers citing this product

1
CSF1/CSF1R Signaling Inhibitor Pexidartinib (PLX3397) Reprograms Tumor-Associated Macrophages and Stimulates T-cell Infiltration in the Sarcoma Microenvironment
Molecular Cancer Therapeutics·2021·258 cit.·DOI
2
The viral protein corona directs viral pathogenesis and amyloid aggregation
Nature Communications·2019·217 cit.·DOI
3
p38 MAP Kinase Mediates Apoptosis through Phosphorylation of BimEL at Ser-65
Journal of Biological Chemistry·2006·207 cit.·DOI
4
Inhibition of Cell Proliferation and Cell Cycle Progression by Specific Inhibition of Basal JNK Activity
Journal of Biological Chemistry·2004·144 cit.·DOI
5
Association between Mitotic Spindle Checkpoint Impairment and Susceptibility to the Induction of Apoptosis by Anti-Microtubule Agents in Human Lung Cancers
American Journal Of Pathology·2003·112 cit.·DOI
6
Ganglioside GM3 inhibits VEGF/VEGFR-2-mediated angiogenesis: Direct interaction of GM3 with VEGFR-2
Glycobiology·2008·105 cit.·DOI
7
The ganglioside GD2 induces the constitutive activation of c-Met in MDA-MB-231 breast cancer cells expressing the GD3 synthase
Glycobiology·2012·105 cit.·DOI
8
NEU1 Sialidase Expressed in Human Airway Epithelia Regulates Epidermal Growth Factor Receptor (EGFR) and MUC1 Protein Signaling
Journal of Biological Chemistry·2012·87 cit.·DOI
9
Genomic Profiling of Messenger RNAs and MicroRNAs Reveals Potential Mechanisms of TWEAK-Induced Skeletal Muscle Wasting in Mice
PLoS ONE·2010·87 cit.·DOI
10
Impaired autophagic and mitochondrial functions are partially restored by ERT in Gaucher and Fabry diseases
PLoS ONE·2019·78 cit.·DOI

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