Cell Signaling Technology

PARP2 (F3Z6Z) Rabbit Monoclonal Antibody

#20009 · $161

100
Citing papers
5.7k
Combined citations
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Research areas

Kruppel-like factors research26Cancer Cells and Metastasis14Cancer-related gene regulation11Genetic Syndromes and Imprinting10Epigenetics and DNA Methylation8Autophagy in Disease and Therapy7Endoplasmic Reticulum Stress and Disease7Chemokine receptors and signaling6

Papers citing this product

1
Cancer-associated fibroblasts induce epithelial–mesenchymal transition of breast cancer cells through paracrine TGF-β signalling
British Journal of Cancer·2013·675 cit.·DOI
2
Cancer-associated fibroblasts regulate the plasticity of lung cancer stemness via paracrine signalling
Nature Communications·2014·441 cit.·DOI
3
MiR-26b, Upregulated in Alzheimer's Disease, Activates Cell Cycle Entry, Tau-Phosphorylation, and Apoptosis in Postmitotic Neurons
Journal of Neuroscience·2013·291 cit.·DOI
4
Melanoma cell-secreted exosomal miR-155-5p induce proangiogenic switch of cancer-associated fibroblasts via SOCS1/JAK2/STAT3 signaling pathway
Journal of Experimental & Clinical Cancer Research·2018·290 cit.·DOI
5
Cancer-associated fibroblasts enhance metastatic potential of lung cancer cells through IL-6/STAT3 signaling pathway
Oncotarget·2017·221 cit.·DOI
6
Axonal transport and secretion of fibrillar forms of α-synuclein, Aβ42 peptide and HTTExon 1
Acta Neuropathologica·2016·158 cit.·DOI
7
Cancer-associated fibroblasts require proline synthesis by PYCR1 for the deposition of pro-tumorigenic extracellular matrix
Nature Metabolism·2022·148 cit.·DOI
8
Regulation of the inflammatory profile of stromal cells in human breast cancer: prominent roles for TNF-α and the NF-κB pathway
Stem Cell Research & Therapy·2015·146 cit.·DOI
9
KLF4 is regulated by RAS/RAF/MEK/ERK signaling through E2F1 and promotes melanoma cell growth
Oncogene·2017·130 cit.·DOI
10
Aberrant low expression of p85α in stromal fibroblasts promotes breast cancer cell metastasis through exosome-mediated paracrine Wnt10b
Oncogene·2017·122 cit.·DOI

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