Cell Signaling Technology

Syk (D3Z1E) Rabbit Monoclonal Antibody

#13198 · $161,385

66
Evidence Score · Well-Established
Used by 502 independent labs across 578 papers. How we score this →
100
Citing papers
4.6k
Combined citations
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Research areas

Phagocytosis and Immune Regulation8Cell Adhesion Molecules Research7Cellular Mechanics and Interactions7Mast cells and histamine7Cell death mechanisms and regulation6Neuroinflammation and Neurodegeneration Mechanisms6DNA Repair Mechanisms5MicroRNA in disease regulation5

Papers citing this product

1
Microglial TREM-1 receptor mediates neuroinflammatory injury via interaction with SYK in experimental ischemic stroke
Cell Death and Disease·2019·296 cit.·DOI
2
Siglec1 suppresses antiviral innate immune response by inducing TBK1 degradation via the ubiquitin ligase TRIM27
Cell Research·2015·191 cit.·DOI
3
H-Ras-specific Activation of Rac-MKK3/6-p38 Pathway
Journal of Biological Chemistry·2005·186 cit.·DOI
4
Hem-1 Complexes Are Essential for Rac Activation, Actin Polymerization, and Myosin Regulation during Neutrophil Chemotaxis
PLoS Biology·2006·185 cit.·DOI
5
NF-κB- and C/EBPβ-driven Interleukin-1β Gene Expression and PAK1-mediated Caspase-1 Activation Play Essential Roles in Interleukin-1β Release from Helicobacter pylori Lipopolysaccharide-stimulated Macrophages
Journal of Biological Chemistry·2004·144 cit.·DOI
6
Epidermal Growth Factor Receptor-dependent and -independent Cell-signaling Pathways Originating from the Urokinase Receptor
Journal of Biological Chemistry·2003·128 cit.·DOI
7
The Rap GTPases Regulate B Cell Migration Toward the Chemokine Stromal Cell-Derived Factor-1 (CXCL12): Potential Role for Rap2 in Promoting B Cell Migration
The Journal of Immunology·2002·116 cit.·DOI
8
Poststroke Induction of  -Synuclein Mediates Ischemic Brain Damage
Journal of Neuroscience·2016·113 cit.·DOI
9
uPAR promotes formation of the p130Cas–Crk complex to activate Rac through DOCK180
The Journal of Cell Biology·2008·109 cit.·DOI
10
HCK induces macrophage activation to promote renal inflammation and fibrosis via suppression of autophagy
Nature Communications·2023·103 cit.·DOI

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