Cell Signaling Technology

LRRK2 (D18E12) Rabbit Monoclonal Antibody

#13046 · $161

68
Citing papers
2.7k
Combined citations
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Research areas

Inflammasome and immune disorders9Amyotrophic Lateral Sclerosis Research7Neuroinflammation and Neurodegeneration Mechanisms6Autophagy in Disease and Therapy5Parkinson's Disease Mechanisms and Treatments5interferon and immune responses4Cellular transport and secretion4Lysosomal Storage Disorders Research4

Papers citing this product

1
Minocycline selectively inhibits M1 polarization of microglia
Cell Death and Disease·2013·749 cit.·DOI
2
Genome-wide CRISPR screen identifies host dependency factors for influenza A virus infection
Nature Communications·2020·244 cit.·DOI
3
Astragaloside IV Alleviates Cerebral Ischemia‐Reperfusion Injury through NLRP3 Inflammasome‐Mediated Pyroptosis Inhibition via Activating Nrf2
Oxidative Medicine and Cellular Longevity·2021·124 cit.·DOI
4
Melatonin modulates neonatal brain inflammation through endoplasmic reticulum stress, autophagy, and mi<scp>R</scp>‐34a/silent information regulator 1 pathway
Journal of Pineal Research·2016·123 cit.·DOI
5
Celastrol Inhibits Dopaminergic Neuronal Death of Parkinson’s Disease through Activating Mitophagy
Antioxidants·2019·107 cit.·DOI
6
Autophagy is essential for maintaining the growth of a human (mini-)organ: Evidence from scalp hair follicle organ culture
PLoS Biology·2018·83 cit.·DOI
7
Inflammasome-Induced IL-1β Secretion in Microglia Is Characterized by Delayed Kinetics and Is Only Partially Dependent on Inflammatory Caspases
Journal of Neuroscience·2015·82 cit.·DOI
8
IFNγ triggers a LIGHT-dependent selective death of motoneurons contributing to the non-cell-autonomous effects of mutant SOD1
Cell Death and Differentiation·2010·76 cit.·DOI
9
HB-EGF Enhances Restitution After Intestinal Ischemia/Reperfusion via PI3K/Akt and MEK/ERK1/2 Activation
Gastroenterology·2005·75 cit.·DOI
10
The ubiquitin ligase Smurf2 suppresses TGFβ-induced epithelial–mesenchymal transition in a sumoylation-regulated manner
Cell Death and Differentiation·2015·72 cit.·DOI

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