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Cell Signaling Technology
UTF1 Antibody
#3909 · $317
100
Citing papers
13.2k
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Research areas
Cell death mechanisms and regulation
9
NF-κB Signaling Pathways
8
Immune cells in cancer
7
Autophagy in Disease and Therapy
7
Ubiquitin and proteasome pathways
6
Phagocytosis and Immune Regulation
6
Cancer, Hypoxia, and Metabolism
5
Neuroinflammation and Neurodegeneration Mechanisms
5
Papers citing this product
1
R428, a Selective Small Molecule Inhibitor of Axl Kinase, Blocks Tumor Spread and Prolongs Survival in Models of Metastatic Breast Cancer
Cancer Research
·
2010
·
550 cit.
·
DOI
2
NF-κB Activation Represses Tumor Necrosis Factor-α-induced Autophagy
Journal of Biological Chemistry
·
2006
·
462 cit.
·
DOI
3
Mammalian TAK1 Activates Snf1 Protein Kinase in Yeast and Phosphorylates AMP-activated Protein Kinase in Vitro
Journal of Biological Chemistry
·
2006
·
461 cit.
·
DOI
4
Monocyte and M1 Macrophage-induced Barrier Defect Contributes to Chronic Intestinal Inflammation in IBD
Inflammatory Bowel Diseases
·
2015
·
361 cit.
·
DOI
5
TNF-α-induced ROS production triggering apoptosis is directly linked to Romo1 and Bcl-XL
Cell Death and Differentiation
·
2010
·
337 cit.
·
DOI
6
TYRO3 induces anti–PD-1/PD-L1 therapy resistance by limiting innate immunity and tumoral ferroptosis
Journal of Clinical Investigation
·
2021
·
311 cit.
·
DOI
7
Mesenchymal Stem Cells Secrete Multiple Cytokines That Promote Angiogenesis and Have Contrasting Effects on Chemotaxis and Apoptosis
PLoS ONE
·
2012
·
305 cit.
·
DOI
8
Glycolytic suppression dramatically changes the intracellular metabolic profile of multiple cancer cell lines in a mitochondrial metabolism-dependent manner
Scientific Reports
·
2019
·
283 cit.
·
DOI
9
Simultaneous Induction of Non-Canonical Autophagy and Apoptosis in Cancer Cells by ROS-Dependent ERK and JNK Activation
PLoS ONE
·
2010
·
260 cit.
·
DOI
10
Dexamethasone Represses Signaling through the Mammalian Target of Rapamycin in Muscle Cells by Enhancing Expression of REDD1
Journal of Biological Chemistry
·
2006
·
257 cit.
·
DOI
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