Cell Signaling Technology

Anti-mouse IgG (H+L) (DyLight® 800 4X PEG Conjugate)

#5257 · $115

100
Citing papers
9.8k
Combined citations
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Research areas

MicroRNA in disease regulation8Cancer-related molecular mechanisms research7RNA Research and Splicing7Autophagy in Disease and Therapy6Metabolism, Diabetes, and Cancer6Cell death mechanisms and regulation5Cytokine Signaling Pathways and Interactions5Cancer, Hypoxia, and Metabolism5

Papers citing this product

1
Anti-CS1 humanized monoclonal antibody HuLuc63 inhibits myeloma cell adhesion and induces antibody-dependent cellular cytotoxicity in the bone marrow milieu
Blood·2007·472 cit.·DOI
2
IL-33 can promote survival, adhesion and cytokine production in human mast cells
Laboratory Investigation·2007·367 cit.·DOI
3
The Leucine-rich Repeat Protein LRIG1 Is a Negative Regulator of ErbB Family Receptor Tyrosine Kinases
Journal of Biological Chemistry·2004·238 cit.·DOI
4
A computational model on the modulation of mitogen-activated protein kinase (MAPK) and Akt pathways in heregulin-induced ErbB signalling
Biochemical Journal·2003·237 cit.·DOI
5
Ligand‐dependent responses of the ErbB signaling network: experimental and modeling analyses
Molecular Systems Biology·2007·237 cit.·DOI
6
Novel Small Molecule Inhibitors of 3-Phosphoinositide-dependent Kinase-1
Journal of Biological Chemistry·2005·227 cit.·DOI
7
TNFα Is Required for Late BRB Breakdown in Diabetic Retinopathy, and Its Inhibition Prevents Leukostasis and Protects Vessels and Neurons from Apoptosis
Investigative Ophthalmology & Visual Science·2011·216 cit.·DOI
8
Adiponectin Sensitizes Insulin Signaling by Reducing p70 S6 Kinase-mediated Serine Phosphorylation of IRS-1
Journal of Biological Chemistry·2007·212 cit.·DOI
9
Strand-Specific miR-28-5p and miR-28-3p Have Distinct Effects in Colorectal Cancer Cells
Gastroenterology·2012·201 cit.·DOI
10
Quantitative Transcriptional Control of ErbB Receptor Signaling Undergoes Graded to Biphasic Response for Cell Differentiation
Journal of Biological Chemistry·2006·186 cit.·DOI

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