Cell Signaling Technology

N-Cadherin Antibody

#4061 · $314

100
Citing papers
10.7k
Combined citations
Supplier page Find this product

Research areas

Cancer-related molecular mechanisms research10MicroRNA in disease regulation9Epigenetics and DNA Methylation7Cancer, Hypoxia, and Metabolism7RNA modifications and cancer6Circular RNAs in diseases5Adipose Tissue and Metabolism5Cancer-related Molecular Pathways5

Papers citing this product

1
TRPA1 induced in sensory neurons contributes to cold hyperalgesia after inflammation and nerve injury
Journal of Clinical Investigation·2005·612 cit.·DOI
2
Deficiency in the Nuclear Factor E2-related Factor-2 Transcription Factor Results in Impaired Adipogenesis and Protects against Diet-induced Obesity
Journal of Biological Chemistry·2010·296 cit.·DOI
3
Heterozygous splice mutation in <i>PIK3R1</i> causes human immunodeficiency with lymphoproliferation due to dominant activation of PI3K
The Journal of Experimental Medicine·2014·282 cit.·DOI
4
Proteins of Diverse Function and Subcellular Location Are Lysine Acetylated in Arabidopsis  
PLANT PHYSIOLOGY·2011·257 cit.·DOI
5
Dissecting the role of H3K27 acetylation and methylation in PRC2 mediated control of cellular identity
Nature Communications·2019·253 cit.·DOI
6
A dysregulated bile acid-gut microbiota axis contributes to obesity susceptibility
EBioMedicine·2020·238 cit.·DOI
7
Tobacco smoking induces cardiovascular mitochondrial oxidative stress, promotes endothelial dysfunction, and enhances hypertension
American Journal of Physiology-Heart and Circulatory Physiology·2019·208 cit.·DOI
8
PTPN14 is required for the density-dependent control of YAP1
Genes & Development·2012·197 cit.·DOI
9
Tumor-Stroma-Inflammation Networks Promote Pro-metastatic Chemokines and Aggressiveness Characteristics in Triple-Negative Breast Cancer
Frontiers in Immunology·2019·193 cit.·DOI
10
Ferulic acid exerts antitumor activity and inhibits metastasis in breast cancer cells by regulating epithelial to mesenchymal transition
Oncology Reports·2016·180 cit.·DOI

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