Cell Signaling Technology

Ataxin-1 Antibody

#2177 · $317

100
Citing papers
20.5k
Combined citations
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Research areas

DNA Repair Mechanisms16Autophagy in Disease and Therapy13Alzheimer's disease research and treatments8Immune cells in cancer7Epigenetics and DNA Methylation7PI3K/AKT/mTOR signaling in cancer6Cancer, Hypoxia, and Metabolism5Neuroinflammation and Neurodegeneration Mechanisms5

Papers citing this product

1
Loss of DMP1 causes rickets and osteomalacia and identifies a role for osteocytes in mineral metabolism
Nature Genetics·2006·1.2k cit.·DOI
2
The Mammalian Target of Rapamycin (mTOR) Pathway Regulates Mitochondrial Oxygen Consumption and Oxidative Capacity
Journal of Biological Chemistry·2006·616 cit.·DOI
3
Plasma cells from multiple myeloma patients express B7-H1 (PD-L1) and increase expression after stimulation with IFN-γ and TLR ligands via a MyD88-, TRAF6-, and MEK-dependent pathway
Blood·2007·575 cit.·DOI
4
Phosphorylation of Mammalian Target of Rapamycin (mTOR) at Ser-2448 IsMediated by p70S6 Kinase
Journal of Biological Chemistry·2005·557 cit.·DOI
5
Shifts in macrophage phenotypes and macrophage competition for arginine metabolism affect the severity of muscle pathology in muscular dystrophy
Human Molecular Genetics·2008·510 cit.·DOI
6
The Rad9–Hus1–Rad1 (9–1–1) clamp activates checkpoint signaling via TopBP1
Genes & Development·2007·461 cit.·DOI
7
Global genomic profiling reveals an extensive p53-regulated autophagy program contributing to key p53 responses
Genes & Development·2013·446 cit.·DOI
8
Baicalin ameliorates neuroinflammation-induced depressive-like behavior through inhibition of toll-like receptor 4 expression via the PI3K/AKT/FoxO1 pathway
Journal of Neuroinflammation·2019·407 cit.·DOI
9
Molecular characterization of Legionella pneumophila-induced interleukin-8 expression in T cells
BMC Microbiology·2010·338 cit.·DOI
10
Amelioration of Alzheimer’s disease pathology by mitophagy inducers identified via machine learning and a cross-species workflow
Nature Biomedical Engineering·2022·333 cit.·DOI