Cell Signaling Technology

Max (E6F6Y) Rabbit Monoclonal Antibody

#17471 · western blot · $161,376

19
Citing papers
206
Combined citations
Supplier page Find this product

Research areas

Ubiquitin and proteasome pathways5Cytokine Signaling Pathways and Interactions2Genetic factors in colorectal cancer2Genetic Neurodegenerative Diseases2Mitochondrial Function and Pathology2Cancer Genomics and Diagnostics1Bioinformatics and Genomic Networks1Amyotrophic Lateral Sclerosis Research1

Papers citing this product

1
The extrinsic apoptosis pathway and its prognostic impact in ovarian cancer
Gynecologic Oncology·2009·54 cit.·DOI
2
The Interplay between NF-kappaB and E2F1 Coordinately Regulates Inflammation and Metabolism in Human Cardiac Cells
PLoS ONE·2011·35 cit.·DOI
3
Rapamycin Attenuated Cardiac Hypertrophy Induced by Isoproterenol and Maintained Energy Homeostasis via Inhibiting NF-<i>κ</i>B Activation
Mediators of Inflammation·2014·28 cit.·DOI
4
Genetic Engineering of Bacteriophage K1F with Human Epidermal Growth Factor to Enhance Killing of Intracellular <i>E. coli</i> K1
ACS Synthetic Biology·2023·18 cit.·DOI
5
Myeloid SENP3 deficiency protects mice from diet and age-induced obesity via regulation of YAP1 SUMOylation
Cellular and Molecular Life Sciences·2023·14 cit.·DOI
6
Evolutionarily conserved genetic interactions with budding and fission yeast MutS identify orthologous relationships in mismatch repair-deficient cancer cells
Genome Medicine·2014·13 cit.·DOI
7
SENP2 Promotes VSMC Phenotypic Switching via Myocardin De-SUMOylation
International Journal of Molecular Sciences·2022·13 cit.·DOI
8
Vincamine ameliorates hepatic fibrosis via inhibiting S100A4‐mediated farnesoid X receptor activation: based on liver microenvironment and enterohepatic circulation dependence
British Journal of Pharmacology·2025·8 cit.·DOI
9
PSME4 Degrades Acetylated YAP1 in the Nucleus of Mesenchymal Stem Cells
Pharmaceutics·2022·7 cit.·DOI
10
Structural basis for the human SENP5’s SUMO isoform discrimination
Nature Communications·2025·6 cit.·DOI

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